Plasma from cirrhotic patients induces inotropic changes on cultured rat heart cells

Life Sciences ◽  
1987 ◽  
Vol 41 (19) ◽  
pp. 2177-2184 ◽  
Author(s):  
Lidia E.P. de Mikulic ◽  
Marie-Claude Auclair ◽  
Catherine Vernimmen ◽  
Didier Lebrec ◽  
Esteban Mikulic
Keyword(s):  
Rat Heart ◽  
Heart Cells ◽  
Cirrhotic Patients ◽  
Rat Heart Cells

scholarly journals Characterization of a strong positive cis-acting element of the human beta-myosin heavy chain gene in fetal rat heart cells.

1992 ◽  
Vol 267 (14) ◽  
pp. 9917-9924 ◽  
Author(s):  
I.L. Flink ◽  
J.G. Edwards ◽  
J.J. Bahl ◽  
C.C. Liew ◽  
M Sole ◽  
...  
Keyword(s):  
Heavy Chain ◽  
Rat Heart ◽  
Chain Gene ◽  
Heart Cells ◽  
Heavy Chain Gene ◽  
Myosin Heavy Chain Gene ◽  
Cis Acting ◽  
Fetal Rat ◽  
Rat Heart Cells

scholarly journals EFFECTS OF CORTISOL ON CULTURED RAT HEART CELLS

1973 ◽  
Vol 57 (1) ◽  
pp. 109-116 ◽  
Author(s):  
J. V. Anastasia ◽  
R. L. McCarl
Keyword(s):  
Fatty Acids ◽  
Fatty Acid ◽  
Free Fatty Acids ◽  
Fatty Acid Oxidation ◽  
Growth Medium ◽  
Rat Heart ◽  
Acid Oxidation ◽  
Heart Cells ◽  
Atp Production ◽  
Rat Heart Cells

This paper reports the determination of the ability of rat heart cells in culture to release [14C]palmitate from its triglyceride and to oxidize this fatty acid and free [14C]palmitate to 14CO2 when the cells are actively beating and when they stop beating after aging in culture. In addition, the levels of glucose, glycogen, and ATP were determined to relate the concentration of these metabolites with beating and with cessation of beating. When young rat heart cells in culture are actively beating, they oxidize free fatty acids at a rate parallel with cellular ATP production. Both fatty acid oxidation and ATP production remain constant while the cells continue to beat. Furthermore, glucose is removed from the growth medium by the cells and stored as glycogen. When cultured cells stop beating, a decrease is seen in their ability to oxidize free fatty acids and to release them from their corresponding triglycerides. Concomitant with decreased fatty acid oxidation is a decrease in cellular levels of ATP until beating ceases. Midway between initiation of cultures and cessation of beating the cells begin to mobilize the stored glycogen. When the growth medium is supplemented with cortisol acetate and given to cultures which have ceased to beat, reinitiation of beating occurs. Furthermore, all decreases previously observed in ATP levels, fatty acid oxidation, and esterase activity are restored.

scholarly journals Distribution of metabolites between mitochondria and cytosol of cultured fibroblastoid rat heart cells

FEBS Letters ◽  
1979 ◽  
Vol 100 (1) ◽  
pp. 125-128 ◽  
Author(s):  
S. Soboll ◽  
K. Werdan ◽  
M. Bozsik ◽  
M. Müller ◽  
E. Erdmann ◽  
...  
Keyword(s):  
Rat Heart ◽  
Heart Cells ◽  
Rat Heart Cells

Calcium Metabolism in Enzymatically Dissociated Rat Heart Cells and in Intact Perfused Ferret Hearts

2020 ◽  
pp. 421-435
Author(s):  
Eduardo Marban ◽  
Shawn W. Robinson ◽  
W. Gil Wier ◽  
Masafumi Kitakaze ◽  
Martin M. Pike ◽  
...  
Keyword(s):  
Rat Heart ◽  
Calcium Metabolism ◽  
Heart Cells ◽  
Rat Heart Cells

Mitochondrial respiratory enzymes are a major target of iron toxicity in rat heart cells

1998 ◽  
Vol 131 (5) ◽  
pp. 466-474 ◽  
Author(s):  
Gabriela Link ◽  
Ann Saada ◽  
Arie Pinson ◽  
Abraham M. Konijn ◽  
Chaim Hershko
Keyword(s):  
Rat Heart ◽  
Iron Toxicity ◽  
Heart Cells ◽  
Respiratory Enzymes ◽  
Rat Heart Cells ◽  
Major Target ◽  
Mitochondrial Respiratory

Accumulation of Taurine by Isolated Rat Heart Cells and Rat Heart Slices

1981 ◽  
pp. 247-259 ◽  
Author(s):  
J. Bahl ◽  
C. J. Frangakis ◽  
B. Larsen ◽  
S. Chang ◽  
D. Grosso ◽  
...  
Keyword(s):  
Rat Heart ◽  
Isolated Rat Heart ◽  
Heart Cells ◽  
Rat Heart Cells ◽  
Isolated Rat
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