scholarly journals B-PO03-009 DUAL AV NODE PATHWAYS MASQUERADING AS SYMPTOMATIC ATRIOVENTRICULAR CONDUCTION DISEASE: THE IMPORTANCE OF TREADMILL STRESS TESTING IN ESTABLISHING SYMPTOM-ARRHYTHMIA CORRELATION

Heart Rhythm ◽  
2021 ◽  
Vol 18 (8) ◽  
pp. S192
Author(s):  
Monica Pammer Austin ◽  
Christopher L. Baldi ◽  
Laura Dennin ◽  
Devan Davis ◽  
Lauren Waronker ◽  
...  
Keyword(s):  
Stress Testing ◽  
Atrioventricular Conduction ◽  
Av Node ◽  
Treadmill Stress

Comparison of changes in R-wave amplitude (RWA) and ST segments in treadmill stress testing (TST) as a predictor of CAD

1978 ◽  
Vol 41 (2) ◽  
pp. 376 ◽  
Author(s):  
George W. Christison ◽  
Peter E. Bonoris ◽  
Paul S. Greenberg ◽  
Mark J. Castellanet ◽  
Myrvin H. Ellestad
Keyword(s):  
Wave Amplitude ◽  
Stress Testing ◽  
St Segments ◽  
R Wave Amplitude ◽  
Treadmill Stress

Separate parasympathetic control of heart rate and atrioventricular conduction of dogs

1990 ◽  
Vol 259 (2) ◽  
pp. H536-H542 ◽  
Author(s):  
D. W. Wallick ◽  
P. J. Martin
Keyword(s):  
Vena Cava ◽  
Atrioventricular Conduction ◽  
Conduction Time ◽  
Fat Pad ◽  
Av Node ◽  
Parasympathetic Ganglia ◽  
Anesthetized Dogs ◽  
Parasympathetic Control ◽  
The Right ◽  
Electrical Stimuli

In open-chest, autonomically decentralized, anesthetized dogs, a brief burst of electrical stimuli was delivered at various time delays to the right pulmonary vein (RPV) fat pad. This fat pad contains parasympathetic ganglia that innervate the sinoatrial (SA) node. Each burst elicited a bimodal increase in the cardiac cycle length (CCL) without eliciting a significant change in atrioventricular conduction time (AVCT). A similar burst was applied to the inferior vena cava-inferior left atrial fat pad. This fat pad contains nerves that innervate the AV node. This latter stimulation elicited a bimodal increase in AVCT without eliciting any change in the CCL. When the cervical vagi were stimulated in a similar manner, a bimodal increase in the CCL was elicited that was similar to the response we observed when the RPV fat pad was stimulated. In contrast, the dromotropic response was quite variable. In conclusion, we could, for the most part, elicit selective parasympathetic control of either the SA or the AV node, respectively.

scholarly journals Echocardiographic assessment of pulmonary artery systolic pressure following treadmill stress testing

2016 ◽  
Vol 18 (11) ◽  
pp. 1278-1282 ◽  
Author(s):  
Arshad A. Khan ◽  
Mohammad Al-Omary ◽  
Ian Renner ◽  
Ehtesham Ul Haque ◽  
Avedis Ekmejian ◽  
...  
Keyword(s):  
Pulmonary Artery ◽  
Stress Testing ◽  
Systolic Pressure ◽  
Pulmonary Artery Systolic Pressure ◽  
Echocardiographic Assessment ◽  
Treadmill Stress

Effect of disopyramide and disobutamide on conduction in perfused rabbit hearts

1981 ◽  
Vol 59 (11) ◽  
pp. 1192-1195
Author(s):  
Peter E. Dresel ◽  
Keith D. Cameron
Keyword(s):  
Antiarrhythmic Agent ◽  
Conduction System ◽  
Atrioventricular Conduction ◽  
Cardiac Conduction ◽  
Conduction Time ◽  
Related Effect ◽  
Av Node ◽  
His Bundle ◽  
Time Changes ◽  
His Bundle Recording

The effects of disopyramide (DP) and a new antiarrhythmic agent, disobutamide (DB) on cardiac conduction were studied using His bundle recording from modified rabbit Langendorff preparations electrically driven at 3 and 4 Hz. Both disopyramide (4–16 μg/mL) and disobutamide (1–30 μg/ml) slowed conduction throughout the atrioventricular conduction system, i.e., SA, AH, and HV intervals were increased in a dose-related manner. Conversion of the conduction time changes to percent changes indicates that disobutamide has a relatively equal effect on each part of the system whereas disopyramide exhibited significantly less effect on AV nodal conduction. Slowing of conduction in the AV node by DP was clearly related to rate. Changes in SA and HV intervals were rate related to a lesser degree. No such rate-related effect was evident with disobutamide. Block of atrial conduction occurred in two out of six hearts when the rate was increased at 8 μg/mL of DP and in three additional hearts at 16 μg/mL. This was interpreted to indicate a change in atrial excitability such that 2 × threshold currents no longer excited the tissues. This was not observed at any concentration of DB.

Blood Pressure Analysis During Treadmill Stress Testing

1981 ◽  
Vol 6 (4) ◽  
pp. 313
Author(s):  
D. L. STONER ◽  
T. H. ALEXANDER ◽  
T. H. ALEXANDER
Keyword(s):  
Blood Pressure ◽  
Stress Testing ◽  
Treadmill Stress ◽  
Pressure Analysis

Depression symptoms predict heart rate recovery after treadmill stress testing

2006 ◽  
Vol 151 (5) ◽  
pp. 1122.e1-1122.e6 ◽  
Author(s):  
Joel W. Hughes ◽  
Elizabeth Casey ◽  
Faith Luyster ◽  
Vicki H. Doe ◽  
Donna Waechter ◽  
...  
Keyword(s):  
Heart Rate ◽  
Stress Testing ◽  
Heart Rate Recovery ◽  
Depression Symptoms ◽  
Treadmill Stress
Sign in / Sign up

Export Citation Format

Share Document