scholarly journals Left Phrenic Nerve Stimulation Due to Breakage of the Endocardial Right Ventricular Lead at the Costoclavicular Ligament

2007 ◽  
Vol 23 (3) ◽  
pp. 250-254
Author(s):  
Mariko Fujimori ◽  
Akira Sugimoto ◽  
Takayuki Shimizu ◽  
Takashi Tamura
2011 ◽  
Vol 5 ◽  
pp. CMC.S6759 ◽  
Author(s):  
Mariana S. Parahuleva ◽  
Ritvan Chasan ◽  
Nedim Soydan ◽  
Yasser Abdallah ◽  
Christiane Neuhof ◽  
...  

Effective cardiac resynchronization therapy (CRT) requires an accurate atrio-biventricular pacing system. The innovative Quartet lead is a quadripolar, over-the-wire left ventricular lead with four electrodes and has recently been designed to provide more options and greater control in pacing vector selection. A lead with multiple pacing electrodes is a potential alternative to physical adjustment of the lead and may help to overcome high thresholds and phrenic nerve stimulation (PNS).


2016 ◽  
Vol 57 (1) ◽  
pp. 118-120 ◽  
Author(s):  
Tomoyuki Kabutoya ◽  
Yasushi Imai ◽  
Hiroaki Watanabe ◽  
Tomonori Watanabe ◽  
Takahiro Komori ◽  
...  

2020 ◽  
Vol 31 (12) ◽  
pp. 3330-3333
Author(s):  
Takayuki Sekihara ◽  
Shinsuke Miyazaki ◽  
Tomokazu Ishida ◽  
Moeko Nagao ◽  
Shota Kakehashi ◽  
...  

2017 ◽  
pp. 247-249
Author(s):  
Daniel D. DuBose

We present a case of a 76-year-old female who developed recurrent left-sided muscle spasms resembling hiccups after permanent dorsal column stimulator (DCS) implantation. The patient had a cardiac resynchronization device with defibrillating capabilities (CRT-D) in place, which was interrogated before and after the permanent DCS placement with no interference reported. Due to the timing of the event with the placement of the DCS, it was presumed that the spasms were related to the DCS implantation, and removal of the DCS was considered. However, further evaluation by a cardiology consultant revealed that a lead from her CRT-D was most likely stimulating the phrenic nerve and causing diaphragmatic contractions. The patient was sent to the electrophysiology clinic where the voltage on her left ventricular lead was reduced, and her symptoms resolved completely. Due to the time, risks, and expense of implanting a DCS, it is imperative to consider all other possible causes of diaphragmatic contractions prior to removing a DCS system. Key words: Dorsal column stimulator, cardiac resynchronization therapy device, phrenic nerve stimulation, hiccups, muscle spasms, diaphragmatic contractions, interference


1989 ◽  
Vol 67 (4) ◽  
pp. 1364-1370 ◽  
Author(s):  
D. F. Speck

Neuronal recordings, microstimulation, and electrolytic and chemical lesions were used to examine the involvement of the Botzinger Complex (BotC) in the bilateral phrenic-to-phrenic inhibitory reflex. Experiments were conducted in decerebrate cats that were paralyzed, ventilated, thoracotomized, and vagotomized. Microelectrode recordings within the BotC region revealed that some neurons were activated by phrenic nerve stimulation (15 of 69 expiratory units, 9 of 67 inspiratory units, and 19 nonrespiratory-modulated units) at average latencies similar to the onset latency of the phrenic-to-phrenic inhibition. In addition, microstimulation within the BotC caused a short latency transient inhibition of phrenic motor activity. In 17 cats phrenic neurogram responses to threshold and supramaximal (15 mA) stimulation of phrenic nerve afferents were recorded before and after electrolytic BotC lesions. In 15 animals the inhibitory reflex was attenuated by bilateral lesions. Because lesion of either BotC neurons or axons of passage could account for this attenuation, in eight experiments the phrenic-to-phrenic inhibitory responses were recorded before and after bilateral injections of 5 microM kainic acid (30–150 nl) into the BotC. After chemical lesions, the inhibitory response to phrenic nerve stimulation remained; however, neuronal activity typical of the BotC could not be located. These results suggest that axons important in producing the phrenic-to-phrenic reflex pass through the region of the BotC, but that BotC neurons themselves are not necessary for this reflex.


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