Demyelination of Nerve Fibers in the Central Nervous System Caused by Chronic Exposure to Natural Hydrogen Sulfide-Containing Gas

2003 ◽  
Vol 136 (4) ◽  
pp. 328-332 ◽  
Author(s):  
T. G. Solnyshkova
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Hydrogen Sulfide ◽  
Chronic Exposure ◽  
Nerve Fibers ◽  
The Central Nervous System

Understanding Neuropathic Pain

CNS Spectrums ◽  
2005 ◽  
Vol 10 (4) ◽  
pp. 298-308 ◽  
Author(s):  
Walter Zieglgänsberger ◽  
Achim Berthele ◽  
Thomas R. Tölle
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Neuropathic Pain ◽  
Neuronal Excitability ◽  
Traumatic Injury ◽  
Nerve Fibers ◽  
Cellular Events ◽  
Excitatory Neurotransmitters ◽  
The Central Nervous System ◽  
Activity Dependent

AbstractNeuropathic pain is defined as a chronic pain condition that occurs or persists after a primary lesion or dysfunction of the peripheral or central nervous system. Traumatic injury of peripheral nerves also increases the excitability of nociceptors in and around nerve trunks and involves components released from nerve terminals (neurogenic inflammation) and immunological and vascular components from cells resident within or recruited into the affected area. Action potentials generated in nociceptors and injured nerve fibers release excitatory neurotransmitters at their synaptic terminals such as L-glutamate and substance P and trigger cellular events in the central nervous system that extend over different time frames. Short-term alterations of neuronal excitability, reflected for example in rapid changes of neuronal discharge activity, are sensitive to conventional analgesics, and do not commonly involve alterations in activity-dependent gene expression. Novel compounds and new regimens for drug treatment to influence activity-dependent long-term changes in pain transducing and suppressive systems (pain matrix) are emerging.

Low concentrations of hydrogen sulphide alter monoamine levels in the developing rat central nervous system

1992 ◽  
Vol 70 (11) ◽  
pp. 1515-1518 ◽  
Author(s):  
B. Skrajny ◽  
R. S. Hannah ◽  
S. H. Roth
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Frontal Cortex ◽  
Hydrogen Sulphide ◽  
Chronic Exposure ◽  
Brain Regions ◽  
Target Organs ◽  
Low Concentrations ◽  
The Central Nervous System ◽  
Developing Rat

The central nervous system is one of the primary target organs for hydrogen sulphide (H2S) toxicity; however, there are limited data on the neurotoxic effects of low-dose chronic exposure on the developing nervous system. Levels of serotonin and norepinephrine in the developing rat cerebellum and frontal cortex were determined following chronic exposure to 20 and 75 ppm H2S during perinatal development. Both monoamines were altered in rats exposed to 75 ppm H2S compared with controls; serotonin levels were significantly increased at days 14 and 21 postnatal in both brain regions, and norepinephrine levels were significantly increased at days 7, 14, and 21 postnatal in cerebellum and at day 21 in the frontal cortex. Exposure to 20 ppm H2S significantly increased the levels of serotonin in the frontal cortex at day 21, whereas levels of norepinephrine were significantly reduced in the frontal cortex at days 14 and 21, and at day 14 in the cerebellum.Key words: hydrogen sulphide, monoamines, serotonin, norepinephrine, neurotoxicity.

scholarly journals Schwannoma of the arythenoid

2013 ◽  
Vol 11 (2) ◽  
pp. 224-226 ◽  
Author(s):  
Carlos Eduardo Molinari Nardi ◽  
Alexandre Wakil Burzichelli ◽  
Elio Gilberto Pfuetzenreiter ◽  
Rogerio Aparecido Dedivitis
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Schwann Cells ◽  
Rare Case ◽  
Nerve Fibers ◽  
Endoscopic Procedure ◽  
The Central Nervous System

Schwannoma is a benign encapsulated tumor that originates from the Schwann cells lining nerve fibers outside the central nervous system. We report a rare case of schwannoma that arose from the left arythenoid cartilage The patient underwent excision of the mass through microlaryngeal endoscopic procedure. No recurrence was observed during follow-up.

MATHEMATIC SIMULATION OF BIOELECTRIC HIP PROSTHESIS MOVEMENT

2017 ◽  
pp. 7-13
Author(s):  
Aleksey Anatolivich Kulik
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Hip Prosthesis ◽  
Control Device ◽  
Nerve Fibers ◽  
Primary Data ◽  
The Central Nervous System ◽  
Physical Abilities ◽  
Prosthesis Control ◽  
And Control

The purpose of the study was to develop a mathematical mechanism which could describe laws of changing electrical signals in nerve fibers in man’s lower extremities and hip prosthesis movement. The article presents a schematic diagram of the bioelectric hip prosthesis control system, main elements of which are an actuator, a control device, and a primary data unit. There are given actual mathematical models of prostheses movement and control signals of man’s central nervous system; on their base was designed the mathematical model controlling bioelectric hip prosthesis movement. A specific feature of the model offered is that the model has a function to characterize a signal transfer from the central nervous system to the actuator of prosthesis. The research results can be applied in the course of hardware and software implementation of bioelectrical prostheses, taking into account physical abilities of the users, creating specific algorithms, studying electric and dynamic characteristics of these devices.

A Suspected New Storage Disease in Cattle

1982 ◽  
Vol 19 (6) ◽  
pp. 616-622 ◽  
Author(s):  
W. J. Hartley ◽  
R. F. Webb
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Brain Stem ◽  
Storage Disease ◽  
Nerve Fibers ◽  
Myelinated Nerve ◽  
Myelinated Nerve Fibers ◽  
The Central Nervous System ◽  
Motor Columns ◽  
The Brain

A suspected storage disease occurred in 50% of a group of five- to seven-month-old Hereford calves in one of four years following the same mating procedure. Lesions were confined to the central nervous system and consisted of multiple intraneuronal, cytoplasmic laminated cytosomes in restricted areas of the brain stem, together with extensive loss of myelinated nerve fibers in the motor columns of the cord.

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