Current advances in the management of cluster headaches

Author(s):  
Theodoros Mavridis ◽  
Marianthi Breza ◽  
Christina Deligianni ◽  
Dimos D. Mitsikostas
Keyword(s):  
Nature ◽  
1999 ◽  
Author(s):  
Sara Abdulla

1983 ◽  
Vol 1 (4) ◽  
pp. 973-996 ◽  
Author(s):  
James J. Corbett
Keyword(s):  

2021 ◽  
Vol 9 ◽  
pp. 2050313X2110236
Author(s):  
Kimberley Yu ◽  
Madeline Chadehumbe

While cluster headaches are classified and considered a primary headache disorder, secondary causes of cluster headaches have been reported and may provide insight into cluster headaches’ potential pathophysiology. The mechanisms underlying this headache phenotype are poorly understood, and several theories have been proposed that range from the activation within the posterior hypothalamus to autonomic tone dysfunction. We provide a review of reported cases in the literature describing secondary causes after cardiac procedures. We will present a novel pediatric case report of a 16-year-old boy with an isolated innominate artery who presented with acute new-onset headaches 8 h following cardiac catheterization of the aortic arch with arteriography and left pulmonary artery stent placement. The headaches were characterized by attacks of excruciating pain behind the left eye and jaw associated with ipsilateral photophobia, conjunctival injection, rhinorrhea, with severe agitation and restlessness. These met the International Classification of Headache Disorders-3 criteria for episodic cluster headaches. The headaches failed to respond to non-steroidal anti-inflammatory medications, dopamine antagonists, and steroids. He showed an immediate response to treatment with oxygen. This unique case of cluster headaches following cardiac catheterization in a pediatric patient with an isolated innominate artery may provide new insight into cluster headaches’ pathogenesis. We hypothesize that the cardiac catheterization induced cardiac autonomic changes that contributed to the development of his cluster headaches. The role of aortic arch anomalies and procedures in potential disruption of the autonomic tone and the causation of cluster headaches is an area requiring further study.


Author(s):  
Susan O’Connell ◽  
Megan Dale ◽  
Helen Morgan ◽  
Kimberley Carter ◽  
Rhys Morris ◽  
...  

2005 ◽  
Vol 11 (4) ◽  
pp. 255-256
Author(s):  
Catherine A. Kernich
Keyword(s):  

Pain Medicine ◽  
2017 ◽  
pp. 527-529
Author(s):  
Paul Rizzoli
Keyword(s):  

2018 ◽  
Vol 32 (1) ◽  
pp. 12-15 ◽  
Author(s):  
Asitha D. L. Jayawardena ◽  
Rakesh Chandra

“Sinus headache” is a common chief complaint that often leads patients to an otolaryngologist's office. Because facial pain may or may not be sinogenic in origin, the otolaryngologist should be equipped to evaluate and treat or to appropriately refer these patients. Analysis of current data indicates that the majority of patients who present with sinus headaches actually have migraines. Furthermore, the downstream effect of the cytokine cascade initiated in migraine physiology can cause rhinologic symptoms, including rhinorrhea, congestion, and lacrimation, which may also confound diagnosis. Other causes of sinus headache include the following: cluster headaches, Sluder neuralgia, trigeminal neuralgia, myofascial trigger point pain (tension headaches, temporomandibular joint dysfunction), and contact point headaches. The diagnostic dilemma for an otolaryngologist occurs when a patient has facial pain and symptoms that may indicate chronic rhinosinusitis but with nondiagnostic endoscopy. Traditionally, these patients have been primarily managed with empiric antibiotics. An alternative strategy is to first screen these patients with an upfront computed tomography. This algorithm may ultimately decrease cost; avert unnecessary antibiotics prescriptions; and prompt more timely referrals to other, more appropriate, disciplines, such as neurology, dentistry, and/or pain management specialists.


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