Prospective Analysis of the Outcome of Subpectoral Breast Augmentation: Sensory Changes, Muscle Function, and Body Image

2004 ◽  
Vol 113 (2) ◽  
pp. 701-707 ◽  
Author(s):  
Jillian Banbury ◽  
Randall Yetman ◽  
Armand Lucas ◽  
Francis Papay ◽  
Karen Graves ◽  
...  
2014 ◽  
Vol 134 ◽  
pp. 92
Author(s):  
Amy Alderman ◽  
Andrea Pusic ◽  
Diane Murphy

2016 ◽  
Vol 137 (6) ◽  
pp. 954e-960e ◽  
Author(s):  
Amy Alderman ◽  
Andrea Pusic ◽  
Diane K. Murphy

2010 ◽  
Vol 1 (2) ◽  
pp. 75-81 ◽  
Author(s):  
Torill Kaasa ◽  
Luis Romundstad ◽  
Helge Roald ◽  
Knut Skolleborg ◽  
Audun Stubhaug

AbstractIn this long-term follow-up study of 175 women, we investigated the prevalence of and factors associated with persisting pain and sensory changes four years after augmentation mammoplasty. Previously the women had participated in an acute postoperative pain study, and follow-up investigations at 6 weeks and 1 year after surgery. In the present study, the women were mailed questionnaires about pain, sensory changes, and affection of daily life, quality of life and pain catastrophizing 4 years after surgery.One hundred and sixteen women answered the questionnaire. The fraction of women reporting evoked- and/or spontaneous pain during the last 24 h had declined from 20% at 1 year to 14% at 4 years. Hyperesthesia had declined from 46% at 1 year to 32% at 4 years, while the change in hypoesthesia was small, 47% at 1 year to 51% at 4 years. Methylprednisolone and parecoxib given pre incisionally reduced acute postoperative pain and reduced the prevalence of hyperesthesia after 6 weeks/1 year, but after 4 years we found no significant differences between the test drug groups. Those having concomitant pain and hyperesthesia at 6 weeks and 1 year had high odds for persisting pain at 4 years (OR 7.8, 95% CI 2.1–29.8, P = 0.003; OR 13.2, 95% CI 2.5–71.3, P = 0.003). In patients without pain but with hyperesthesia at 1 year, the hyperesthesia increased the odds for pain at 4 years (OR 2.6 95% CI 1.1–6.1, P = 0.03). Hypoesthesia at 6 weeks or at 1 year did not affect the odds for pain at 4 years. A good general health condition (mental and physical) was associated with reduced odds for pain at 4 years (OR = 0.56, 95% CI 0.35–0.88, P = 0.01). However, using the Short Form health survey, SF-12, the Mental Component Summary Score seemed to affect the odds for chronic pain more than the Physical Component Summary Score.To conclude, the prevalence of pain and hyperesthesia after breast augmentation declined from 1 to 4 years. Nevertheless, the most striking finding in the current trial was that pain coinciding with hyperesthesia at 6 weeks and 1 year resulted in highly increased odds for persistent postoperative pain. Even hyperesthesia alone, without pain, increased the odds for chronic postsurgical pain. Thus, the present study suggests hyperesthesia as an independent risk factor for chronic postsurgical pain.


2012 ◽  
Vol 3 (3) ◽  
pp. 195-196
Author(s):  
T. Kaasa ◽  
L. Romundstad ◽  
A. Stubhaug

AbstractBackground/aimsThe mechanisms behind chronic postsurgical pain remains unsettled. Quantitative sensory testing and questioning sensory function may help understand mechanisms behind the transition from acute to chronic pain. The aim of this study was to assess these aspects in postsurgical patients with and without chronic postsurgical pain.Methods116 women, who answered a questionnaire in a four years follow-up study of pain, sensory changes and quality of life after cosmetic breast augmentation surgery [1], were invited to participate in a psychophysical study. Twenty women answered the request and filled in a questionnaire, and 12 of these women finally met for examination.ResultsSix of the 12 women had pain in the area of surgery, three were pain-free, but reported sensory changes and three reported no pain or sensory disturbances. We performed a detailed quantitative sensory examination, with a protocol adapted from Rollke et al. [2] While only 3/6 patients in the pain-free group reported hypoesthesia, an area of hypoesthesia to tactile-, heat- and cold stimuli was identified in all subjects when examined (Table 1).Table 1Number in each group (subjects with pain vs. subjects without pain) showing presence of sensory characteristics.Pain (N = 6)No pain (N = 6)QuestionnaireHypoesthesia53Hyperesthesia63ExaminationHypoesthesia (tactile, heat, cold)66Hypersensitivity detected41Hyperpathia to heat55Paradoxical heat sensation during cold stimulation40Allodynia, cotton00Allodynia, brush01Cold allodynia51Abnormal temporal summation51Deep pain after algometry40The most striking difference between the group reporting pain and the one not reporting pain was the presence of paradoxical heat sensation, cold allodynia, abnormal temporal summation, and the presence of deep pain during/after pressure pain threshold testing with the algometer in the group with pain.ConclusionSelf-reported sensory changes under-estimated sensory changes. Sensory testing revealed signs of peripheral nerve injury changes in all subjects, while signs of central nervous changes were found predominantly in patients with persistent pain.


2011 ◽  
Vol 15 (3) ◽  
pp. 328-332 ◽  
Author(s):  
Marie L Sperlingl ◽  
Hilde Høimyrl ◽  
Kenneth Finnerupl ◽  
Troels S Jensenl ◽  
Nanna B Finnerupl

1998 ◽  
Vol 101 (7) ◽  
pp. 1956-1961 ◽  
Author(s):  
David B. Sarwer ◽  
Scott P. Bartlett ◽  
Louis P. Bucky ◽  
Don LaRossa ◽  
David W. Low ◽  
...  

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