Succinate oxidation by coupled potato tuber mitochondria followed by rapid scan spectrometry

1980 ◽  
Vol 50 (3) ◽  
pp. 241-250 ◽  
Author(s):  
GASTON DUCET
Keyword(s):  
Potato Tuber ◽  
Succinate Oxidation ◽  
Rapid Scan

Immunolocalization of extensin and potato tuber lectin in carrot, tomato and potato

1996 ◽  
Vol 97 (4) ◽  
pp. 708-718 ◽  
Author(s):  
Shu-xia Li ◽  
Allan M. Showalter
Keyword(s):  
Potato Tuber

TRIAL UTILIZATION OF RAPID SCAN OBSERVATION OF HIMAWARI-8 FOR OBTAINING INFORMATION ON CUMULUS LIFE STAGE

2018 ◽  
Vol 74 (5) ◽  
pp. I_283-I_288
Author(s):  
Wendi HARJUPA ◽  
Eiichi NAKAKITA ◽  
Yasuhiko SUMIDA ◽  
Aritoshi MASUDA
Keyword(s):  
Life Stage ◽  
Rapid Scan

The Multiple-Vortex Structure of the El Reno, Oklahoma, Tornado on 31 May 2013

2018 ◽  
Vol 146 (8) ◽  
pp. 2483-2502 ◽  
Author(s):  
Howard B. Bluestein ◽  
Kyle J. Thiem ◽  
Jeffrey C. Snyder ◽  
Jana B. Houser
Keyword(s):  
Doppler Radar ◽  
Radar Data ◽  
Rapid Scan ◽  
X Band ◽  
Close Range ◽  
Formation And Evolution ◽  
Using Data ◽  
Secondary Vortices ◽  
The Right ◽  
Gust Front

Abstract This study documents the formation and evolution of secondary vortices associated within a large, violent tornado in Oklahoma based on data from a close-range, mobile, polarimetric, rapid-scan, X-band Doppler radar. Secondary vortices were tracked relative to the parent circulation using data collected every 2 s. It was found that most long-lived vortices (those that could be tracked for ≥15 s) formed within the radius of maximum wind (RMW), mainly in the left-rear quadrant (with respect to parent tornado motion), passing around the center of the parent tornado and dissipating closer to the center in the right-forward and left-forward quadrants. Some secondary vortices persisted for at least 1 min. When a Burgers–Rott vortex is fit to the Doppler radar data, and the vortex is assumed to be axisymmetric, the secondary vortices propagated slowly against the mean azimuthal flow; if the vortex is not assumed to be axisymmetric as a result of a strong rear-flank gust front on one side of it, then the secondary vortices moved along approximately with the wind.

scholarly journals Covalent Structures of Potato Tuber Lipases (Patatins) and Implications for Vacuolar Import

2009 ◽  
Vol 284 (15) ◽  
pp. 9764-9769 ◽  
Author(s):  
Karen G. Welinder ◽  
Malene Jørgensen
Keyword(s):  
Potato Tuber

scholarly journals Release of Apical Dominance in Potato Tuber Is Accompanied by Programmed Cell Death in the Apical Bud Meristem

2012 ◽  
Vol 158 (4) ◽  
pp. 2053-2067 ◽  
Author(s):  
Paula Teper-Bamnolker ◽  
Yossi Buskila ◽  
Yael Lopesco ◽  
Shifra Ben-Dor ◽  
Inbal Saad ◽  
...  
Keyword(s):  
Cell Death ◽  
Programmed Cell Death ◽  
Potato Tuber ◽  
Apical Dominance ◽  
Apical Bud

scholarly journals Stoichiometry of mitochondrial H+ translocation coupled to succinate oxidation at level flow.

1984 ◽  
Vol 259 (8) ◽  
pp. 4802-4811 ◽  
Author(s):  
L E Costa ◽  
B Reynafarje ◽  
A L Lehninger
Keyword(s):  
Succinate Oxidation

Molecular regulation of potato tuber dormancy and sprouting: a mini-review

2021 ◽  
Author(s):  
Hui-Ling Gong ◽  
Leonce Dusengemungu ◽  
Clement Igiraneza ◽  
Placide Rukundo
Keyword(s):  
Potato Tuber ◽  
Molecular Regulation ◽  
Tuber Dormancy

scholarly journals Evidence that metformin exerts its anti-diabetic effects through inhibition of complex 1 of the mitochondrial respiratory chain

2000 ◽  
Vol 348 (3) ◽  
pp. 607-614 ◽  
Author(s):  
Mark R. OWEN ◽  
Elena DORAN ◽  
Andrew P. HALESTRAP
Keyword(s):  
Respiratory Chain ◽  
Rat Hepatocytes ◽  
Mitochondrial Respiratory Chain ◽  
Time Dependent ◽  
Succinate Oxidation ◽  
Isolated Rat Hepatocytes ◽  
Peripheral Tissues ◽  
Dependent Inhibition ◽  
Insulin Dependent ◽  
Mitochondrial Oxidation

Although metformin is widely used for the treatment of non-insulin-dependent diabetes, its mode of action remains unclear. Here we provide evidence that its primary site of action is through a direct inhibition of complex 1 of the respiratory chain. Metformin (50 μM) inhibited mitochondrial oxidation of glutamate+malate in hepatoma cells by 13 and 30% after 24 and 60 h exposure respectively, but succinate oxidation was unaffected. Metformin also caused time-dependent inhibition of complex 1 in isolated mitochondria, whereas in sub-mitochondrial particles inhibition was immediate but required very high metformin concentrations (K0.5, 79 mM). These data are compatible with the slow membrane-potential-driven accumulation of the positively charged drug within the mitochondrial matrix leading to inhibition of complex 1. Metformin inhibition of gluconeogenesis from L-lactate in isolated rat hepatocytes was also time- and concentration-dependent, and accompanied by changes in metabolite levels similar to those induced by other inhibitors of gluconeogenesis acting on complex 1. Freeze-clamped livers from metformin-treated rats exhibited similar changes in metabolite concentrations. We conclude that the drug's pharmacological effects are mediated, at least in part, through a time-dependent, self-limiting inhibition of the respiratory chain that restrains hepatic gluconeogenesis while increasing glucose utilization in peripheral tissues. Lactic acidosis, an occasional side effect, can also be explained in this way.

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