William Rutherford Sanders (1828–1881), anatomist, physician, linguist and museum conservator

2018 ◽  
Vol 28 (2) ◽  
pp. 120-123
Author(s):  
Dugald Gardner

William Rutherford Sanders spent a childhood and early student days divided between Edinburgh and Montpelier, France before graduating in Medicine in Edinburgh. An early interest in the spleen was encouraged by a two-year period in Europe where he became familiar with the work of Helmholtz, Bernard and Henle. Returning to Edinburgh, his growing experience led to the position of assistant in the Infirmary pathology department. He conducted classes in the University of Edinburgh and on behalf of the Royal Colleges became familiar with the museum of the Royal College of Surgeons where he was chosen as Conservator in 1853. Criticised by 20th century historians for concentrating on verbal teaching rather than on the conservation of the museum, Sanders became a consultant physician to the Royal Infirmary in 1861 and in 1869 Professor of General Pathology. Throughout these years, Sanders gave as much time as possible to the study of the structure and function of the spleen and to neurological disorders such as hemiplegia. His later life was interrupted by a series of illnesses commencing with an abdominal abscess. A prolonged convalescence allowed the resumption of work but deranged vision and hemiplegia preceded his death on 18 February 1881.

2021 ◽  
Vol 134 (16) ◽  

ABSTRACT First Person is a series of interviews with the first authors of a selection of papers published in Journal of Cell Science, helping early-career researchers promote themselves alongside their papers. Fanny Jaudon and Martina Albini are co-first authors on ‘ A developmental stage- and Kidins220-dependent switch in astrocyte responsiveness to brain-derived neurotrophic factor’, published in JCS. Fanny is a postdoc at the University of Trieste in the lab of Lorenzo A. Cingolani at Center for Synaptic Neuroscience and Technology, Istituto Italiano di Tecnologia, Genova, Italy, investigating the molecular mechanisms controlling development and function of neuronal circuits and implementing genome-editing approaches for the treatment of neurological disorders. Martina is a PhD student at the Istituto Italiano di Tecnologia in the lab of Fabio Benfenati and Fabrizia Cesca investigating neurotrophin biology and its involvement in neurological diseases.


1937 ◽  
Vol 59 (1) ◽  
pp. 119-164 ◽  
Author(s):  
Vera Fretter

The work described in this paper was carried out in the Department of Zoology of Birkbeck College, University of London, and at the Marine Laboratories at Plymouth and Port Erin. I wish to acknowledge my indebtedness to the University of London for the use of their table at Plymouth, to Birkbeck College for a grant of £5 towards the illustrations, and, in particular, I would express my gratitude to Mr Graham, under whose direction the work was carried out, for his continued kindness and help.The specimens of Lepidochitona cinereus which were used for the investigation were collected at Bangor, Plymouth and Port Erin, and also obtained from Cullercoats. Most of the material from Plymouth and one specimen from Cullercoats was infected with the Haplosporidian parasite Haplosporidium chitonis (Debaisieux, 1920) and was therefore unreliable for histological and physiological work. Specimens of Acanthochitona crinitus were collected at Plymouth, and showed no sign of infection with Haplosporidium chitonis; a few, however, were slightly parasitised with another Sporozoan. The Californian species Ischnochiton magdalenensis and Cryptochiton stelleri were obtained from Turtox General Biological Supply House, Chicago. Specimens of these species were fixed in formalin, which proved to be a poor fixative for histological work.


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