The technique to open de pericardium in uniportal VATS and it also demonstrates the importance of a thorough dissection between the right main pulmonary artery and the right main bronchus, and in the caudal part of the superior pulmonary vein

ASVIDE ◽  
2020 ◽  
Vol 7 ◽  
pp. 33-33
Author(s):  
William Guido-Guerrero ◽  
Diego Gonzalez-Rivas
Keyword(s):  
Pulmonary Artery ◽  
Pulmonary Vein ◽  
Main Bronchus ◽  
Main Pulmonary Artery ◽  
Caudal Part ◽  
Superior Pulmonary Vein ◽  
The Right

Cardiovascular-associated tracheobronchial obstruction in children

2012 ◽  
Vol 23 (2) ◽  
pp. 233-238 ◽  
Author(s):  
Shen-Hao Lai ◽  
Sui-Ling Liao ◽  
Kin-Sun Wong
Keyword(s):  
Pulmonary Artery ◽  
Airway Obstruction ◽  
Main Bronchus ◽  
Main Pulmonary Artery ◽  
Diagnostic Tools ◽  
Radiographic Findings ◽  
Tracheal Compression ◽  
Airway Obstructions ◽  
Right Pulmonary Artery ◽  
The Right

AbstractTracheobronchial compression of cardiovascular origin is an uncommon and frequently unrecognised cause of respiratory distress in children. The compression may be due to encircling vessels or dilated neighbouring cardiovascular structures. Bronchoscopy and detailed radiography, especially computed tomography and magnetic resonance imaging, are among the most powerful diagnostic tools. Few previous reports have addressed the relationship between bronchoscopic findings and underlying cardiovascular anomalies. The objective of this study was to correlate bronchoscopic and radiographic findings in children with cardiovascular-associated airway obstruction. A total of 41 patients were recruited for the study. Patients with airway obstruction were stratified on the basis of the aetiology of the cardiovascular structures and haemodynamics into an anatomy-associated group and a haemodynamics-associated group. In the anatomy-associated group, stenosis and malacia were found with comparable frequency on bronchoscopy, and the airway obstructions were mostly found in the trachea (71% of patients). In the haemodynamics-associated group, malacia was the most common bronchoscopic finding (85% of patients), and nearly all locations of airway involvement were in the airway below the carina (90% of patients). The tracheal compression was usually caused by aberrant systemic branching arteries in the anatomy-associated group. In the haemodynamics-associated group, the causal relationships varied. Tracheal compression was often caused by lesions of the main pulmonary artery and aorta, whereas obstruction of the right main bronchus was caused by lesions of the main pulmonary artery and right pulmonary artery. The causes of left main bronchus compression were more diverse. In summary, the bronchoscopic presentations and locations are quite different between these two groups.

Differential sympathetic regulation of automatic, conductile, and contractile tissue in dog heart

1988 ◽  
Vol 255 (5) ◽  
pp. H1050-H1059 ◽  
Author(s):  
J. L. Ardell ◽  
W. C. Randall ◽  
W. J. Cannon ◽  
D. C. Schmacht ◽  
E. Tasdemiroglu
Keyword(s):  
Pulmonary Artery ◽  
Pulmonary Vein ◽  
Contractile Force ◽  
Right Atrial ◽  
Canine Heart ◽  
Superior Pulmonary Vein ◽  
Right Pulmonary Artery ◽  
The Common ◽  
The Right ◽  
Contractile Tissue

Sympathetic pathways mediating chronotropic, dromotropic, and inotropic responses during ansae subclavia stimulation were determined by sequential dissection around major cardiac vessels. Right sympathetic (RS) projections influencing ventricular contractile force converge at the common pulmonary artery and within the pulmonary artery nerves (PAN). RS projections influencing left atrial contractile force course within the PANs. RS pathways to pacemaker and right atrial contractile tissue were localized between the superior vena cava and ascending aorta. RS projections influencing conductile tissue converge between the common pulmonary artery and proximal right pulmonary artery. Left sympathetic (LS) projections to ventricular contractile tissue were localized at the common pulmonary artery, within the PANs, and in the ventral lateral cardiac nerve (VLCN). LS pathways influencing heart rate and conductile tissue were localized at the left pulmonary artery and coursing between the right pulmonary artery and left superior pulmonary vein. LS projections to atrial contractile tissue were localized within the PANs and coursing between the right pulmonary artery and left superior pulmonary vein. We conclude that there are parallel, yet distinct, projections of sympathetic efferents to automatic, conductile, and contractile tissue of the canine heart.

A pseudo sinus tachycardia originating from the right superior pulmonary vein

2008 ◽  
Vol 63 (2) ◽  
pp. 265-269
Author(s):  
C. Scavée ◽  
A. Brasseur ◽  
R. Weerasooriya
Keyword(s):  
Pulmonary Vein ◽  
Sinus Tachycardia ◽  
Superior Pulmonary Vein ◽  
The Right

scholarly journals A pseudo-sinus rhythm due to bigeminal ectopy with the focus in the right superior pulmonary vein

2018 ◽  
Vol 4 (11) ◽  
pp. 553-555
Author(s):  
Marina Arai ◽  
Seiji Fukamizu ◽  
Rintaro Hojo ◽  
Masayasu Hiraoka
Keyword(s):  
Sinus Rhythm ◽  
Pulmonary Vein ◽  
Superior Pulmonary Vein ◽  
The Right

THE HAMMAN-RICH SYNDROME IN CHILDHOOD

PEDIATRICS ◽  
1958 ◽  
Vol 22 (2) ◽  
pp. 279-288
Author(s):  
Israel Diamond
Keyword(s):  
Pulmonary Artery ◽  
Left Atrium ◽  
Pulmonary Vein ◽  
Clinical Picture ◽  
Lung Biopsy ◽  
Progressive Dyspnea ◽  
Fibrotic Process ◽  
Persistent Cough ◽  
Right Pulmonary Artery ◽  
The Right

The Hamman-Rich syndrome is described in a 4-year-old Negro male. The clinical picture was that of persistent cough and progressive dyspnea beginning at 4 months of age. Diagnosis was made ante mortem by lung biopsy. The fibrotic process and arteriolosclerosis were more marked in the right lung. The disease may have been initiated by a bout of aspiration. There was accompanying stenosis of the right pulmonary artery and vein and occlusion of the lumen of the right pulmonary vein at its entrance to the left atrium. The hilar vascular findings are believed to be secondary to hilar areolar inflammation.

scholarly journals Saddle pulmonary emboli: an unusual presentation

CJEM ◽  
2009 ◽  
Vol 11 (06) ◽  
pp. 558-559
Author(s):  
Truptesh H. Kothari ◽  
Shivangi Kothari ◽  
Mahima Pandey ◽  
Harshit Khara ◽  
Nishant Dhungel
Keyword(s):  
Pulmonary Artery ◽  
Troponin I ◽  
Femoral Vein ◽  
Main Pulmonary Artery ◽  
Vena Cava ◽  
Factor V Leiden ◽  
Superficial Femoral Vein ◽  
Presenting Symptoms ◽  
Coronary Syndrome ◽  
The Right

A 38-year-old man with a history of polyposis syndrome diagnosed 3 years previously, with poor compliance for follow-up, presented to the emergency department with symptoms of retrosternal chest pain associated with dizziness and shortness of breath. His blood pressure was 94/43 mm Hg, his pulse was 123 beats/min and he had an oxygen saturation of 84% on room air. The patient’s initial laboratory results showed a hemoglobin of 80 g/L and blood gas with a pH of 7.23. He had a normal chest radiograph and electrocardiogram, but had an elevated troponin I at 0.12 μg/L. He was given acetylsalicylic acid for suspicion of acute coronary syndrome. On physical examination, the patient was found to have right calf tenderness. With this finding and the presenting symptoms, he underwent computed tomography angiography (CTA) of the chest. The chest CTA showed a massive saddle embolus with a filling defect completely occluding the right pulmonary artery and extending through the main pulmonary artery segment to involve the left pulmonary artery. There were also diffuse filling defects involving bilateral pulmonary segmental arteries (Fig. 1 and Fig. 2). The patient received alteplase and underwent a workup for a hypercoagulable state. His workup revealed positive anticardiolipin antibodies and factor V Leiden. The Doppler ultrasound of his lower extremities showed an extensive thrombus measuring more than 6 cm extending in the right superficial femoral vein. The patient was then referred for placement of an inferior vena cava filter.

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