Multiple cranial nerve palsies due to a hyperextension injury to the cervical spine

1984 ◽  
Vol 61 (1) ◽  
pp. 172-173 ◽  
Author(s):  
Louis Rosa ◽  
Mark Carol ◽  
Roberto Bellegarrigue ◽  
Thomas B. Ducker
Keyword(s):  
Spinal Cord ◽  
Cervical Spine ◽  
Cranial Nerve ◽  
Content Type ◽  
Hyperextension Injury ◽  
Cranial Nerve Palsies ◽  
Multiple Cranial Nerve ◽  
Fine Print

✓ The case of a patient with multiple bilateral cranial nerve palsies and spinal cord sparing secondary to a stable hyperextension injury to C-1 is presented.

Recurrent subarachnoid hemorrhage complicating cerebral arterial ectasia

1981 ◽  
Vol 55 (1) ◽  
pp. 139-142 ◽  
Author(s):  
Steven J. Goldstein ◽  
Phillip A. Tibbs
Keyword(s):  
Subarachnoid Hemorrhage ◽  
Cranial Nerve ◽  
Review Of The Literature ◽  
Radiographic Findings ◽  
Content Type ◽  
Clinical Complications ◽  
Cranial Nerve Palsies ◽  
Arterial Ectasia ◽  
Fine Print

✓ A case of subarachnoid hemorrhage (SAH) complicating cerebral arterial ectasia is reported. While ischemia and cranial nerve palsies are commonly associated with this condition, review of the literature reveals that SAH is exceedingly rare. The pathogenesis, radiographic findings, and clinical complications of cerebral arterial ectasia are discussed.

Transsphenoidal approach to infrasellar tumors involving the cavernous sinus

1990 ◽  
Vol 73 (4) ◽  
pp. 513-517 ◽  
Author(s):  
Nobuo Hashimoto ◽  
Haruhiko Kikuchi
Keyword(s):  
Cavernous Sinus ◽  
Cranial Nerve ◽  
Cranial Nerves ◽  
Transsphenoidal Approach ◽  
Skull Base Tumors ◽  
Content Type ◽  
Operating Field ◽  
Cranial Nerve Palsies ◽  
Operative Complications ◽  
Fine Print

✓ The authors review their 2-year experience with a rhinoseptal transsphenoidal approach to skull-base tumors of various pathologies involving both the sphenoid and cavernous sinuses. Eight patients with cranial nerve palsies attributable to compression of the contents of the cavernous sinus and/or optic canal are included in this report. Among these patients, a total of 17 cranial nerves were affected. Postoperative normalization was achieved in eight nerves, significant improvement in seven nerves, and no improvement in two nerves. There were no operative complications of aggravation of cranial nerve palsies in this series. In spite of the limited operating field, the results demonstrate the effectiveness and safety of this approach. The authors recommend that this approach be considered before more aggressive surgery is undertaken.

Complications of halo-pelvic traction

1976 ◽  
Vol 45 (6) ◽  
pp. 716-718 ◽  
Author(s):  
Rodney A. Rozario ◽  
Bennett M. Stein
Keyword(s):  
Blood Supply ◽  
Cranial Nerve ◽  
Cranial Nerves ◽  
Rapid Rate ◽  
Content Type ◽  
Cranial Nerve Palsies ◽  
Fine Print

✓ When halo-pelvic traction is applied at a rapid rate it may induce cranial nerve palsies. The sixth, ninth, and tenth cranial nerves appear to be the most vulnerable. A proposed etiology is the stretching of these nerves resulting in a compromised blood supply with a consequent temporary paralysis which usually improves within 8 to 10 weeks.

Calcifying pseudoneoplasms of the spine with myelopathy

2000 ◽  
Vol 93 (2) ◽  
pp. 291-293 ◽  
Author(s):  
Matthew T. Mayr ◽  
Stephen Hunter ◽  
Scott C. Erwood ◽  
Regis W. Haid
Keyword(s):  
Spinal Cord ◽  
Cervical Spine ◽  
Thoracic Spine ◽  
Radiological Evaluation ◽  
Pathological Findings ◽  
Content Type ◽  
Progressive Myelopathy ◽  
Fine Print

✓ The authors describe two cases of calcifying pseudoneoplasms, rare degenerative lesions that mimic tumor or infection. One case involved the cervical spine and the second the thoracic spine. Both patients experienced progressive myelopathy from extradural compression of the spinal cord. The radiological evaluation, pathological findings in the lesions, treatment, and follow up are described. Total or subtotal excision can relieve symptoms and prevent recurrence of this lesion.

Congenital neural abnormalities presenting with mirror movements in a patient with Klippel-Feil syndrome

1983 ◽  
Vol 59 (5) ◽  
pp. 891-894 ◽  
Author(s):  
Ian R. Whittle ◽  
Michael Besser
Keyword(s):  
Spinal Cord ◽  
Cervical Spine ◽  
Posterior Fossa ◽  
Pyramidal Tract ◽  
Cervical Spinal Cord ◽  
Content Type ◽  
Mirror Movements ◽  
Posterior Fossa Cyst ◽  
Vertebral Anomalies ◽  
Fine Print

✓ A young girl with Klippel-Feil syndrome presented with the onset of mirror movements in early childhood. Computerized tomography studies of her cervical spine and brain revealed fibrous diastematomyelia with duplication of the cervical spinal cord and an extra-axial midline posterior fossa cyst, together with the multiple cervical vertebral anomalies. Exploration of the posterior fossa lesion revealed it to be a dermoid cyst. The congenital spinal and cord abnormalities found in this case support the hypothesis that the Klippel-Feil syndrome may be associated with variable duplication of the spinal cord and that mirror movements may be related to impairment of pyramidal tract decussation.

Chondroma of the cervical spine

1987 ◽  
Vol 66 (1) ◽  
pp. 128-130 ◽  
Author(s):  
Gérald Lozes ◽  
Ahmad Fawaz ◽  
Harry Perper ◽  
Philippe Devos ◽  
Pascal Benoit ◽  
...  
Keyword(s):  
Spinal Cord ◽  
Cervical Spine ◽  
Spinal Cord Compression ◽  
Vertebral Column ◽  
Cord Compression ◽  
Neurological Recovery ◽  
Surgical Removal ◽  
Content Type ◽  
Fine Print

✓ The authors report a case of cervical chondroma presenting with a syndrome of spinal cord compression in a 76-year-old woman. Total surgical removal of the lesion was followed by partial neurological recovery. Chondromas of the vertebral column are rarely reported in the literature.

Transverse fracture of the clivus

1973 ◽  
Vol 39 (5) ◽  
pp. 610-614 ◽  
Author(s):  
Bob B. Sanders ◽  
Gary D. Vanderark
Keyword(s):  
Cranial Nerve ◽  
Young Patients ◽  
Clinical Findings ◽  
Horner’S Syndrome ◽  
Transverse Fracture ◽  
Radiological Findings ◽  
Horner's Syndrome ◽  
Content Type ◽  
Cranial Nerve Palsies ◽  
Fine Print

✓ Three young patients with transverse fracture of the clivus exhibited clinical findings of progressive cranial nerve palsies associated with a Horner's syndrome. Clinical and radiological findings of this syndrome are described.

Multiple false localizing signs in intracranial tumor

1974 ◽  
Vol 40 (2) ◽  
pp. 264-266 ◽  
Author(s):  
Andrew R. Turnbull
Keyword(s):  
Cranial Nerve ◽  
Intracranial Tumor ◽  
Content Type ◽  
Cerebellar Hemangioblastoma ◽  
Mode Of Presentation ◽  
Cranial Nerve Palsies ◽  
Fine Print

✓ A case of cerebellar hemangioblastoma is presented in which multiple false localizing signs caused difficulty in the clinical localization of the lesion. The mode of presentation and pathogenesis of these signs are briefly discussed, and the observation made that the ninth and tenth cranial nerve palsies recorded in this case rarely lead to false localization.

Anastomotic connections between the eleventh nerve and the posterior root of the first cervical nerve in humans

1973 ◽  
Vol 38 (2) ◽  
pp. 189-197 ◽  
Author(s):  
G. Ouaknine ◽  
H. Nathan
Keyword(s):  
Spinal Cord ◽  
Cranial Nerve ◽  
Spinal Accessory Nerve ◽  
Posterior Root ◽  
Cervical Nerve ◽  
Content Type ◽  
Spinal Accessory ◽  
Points Of View ◽  
Sensory Fibers ◽  
Fine Print

✓ A study of connections between C-1, C-2, and the spinal accessory nerve is reported. Four variations are described from anatomical and clinical points of view. Often the only pathway for the sensory fibers of C-1 to reach the spinal cord is through the rootlets of the eleventh cranial nerve.

Pathogenesis of myelopathy in patients with ossification of the posterior longitudinal ligament

2002 ◽  
Vol 96 (2) ◽  
pp. 168-172 ◽  
Author(s):  
Shunji Matsunaga ◽  
Makoto Kukita ◽  
Kyoji Hayashi ◽  
Reiko Shinkura ◽  
Chihaya Koriyama ◽  
...  
Keyword(s):  
Spinal Cord ◽  
Cervical Spine ◽  
Critical Point ◽  
Clinical Symptoms ◽  
Posterior Longitudinal Ligament ◽  
Static Compression ◽  
Content Type ◽  
Dynamic Factors ◽  
The Relationship ◽  
Fine Print

Object. The goal of this study was to clarify the pathogenesis of myelopathy in patients with ossification of the posterior longitudinal ligament (OPLL) based on the relationship between static compression factors and dynamic factors. Methods. There was a total of 247 patients, including 167 patients who were conservatively followed for a mean of 11 years and 2 months and 80 patients who had myelopathy at initial consultation and underwent surgery. The changes in clinical symptoms associated with OPLL in the cervical spine were examined periodically. During the natural course of OPLL in the cervical spine, 37 (22%) of 167 patients developed or suffered aggravated spinal symptoms. All of the patients with a space available for the spinal cord (SAC) less than 6 mm suffered myelopathy, whereas the patients with an SAC diameter of 14 mm or greater did not. No correlation was found between the presence or absence of myelopathy in patients whose SAC diameter ranged from 6 mm to less than 14 mm. In patients with myelopathy whose minimal SAC diameter ranged from 6 mm to less than 14 mm, the range of motion of the cervical spine was significantly greater. Conclusions. These results indicate that pathological compression by the ossified ligament above a certain critical point may be the most significant factor in inducing myelopathy, whereas below that point dynamic factors may be largely involved in inducing myelopathy.

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