Combined sagittal and lateral sinus dural fistulae occlusion

1995 ◽  
Vol 82 (2) ◽  
pp. 159-165 ◽  
Author(s):  
Sean Mullan ◽  
Douglas L. Johnson

✓ The clinical course of three patients who had combined sagittal and lateral sinus dural fistulae is described. One patient, with impending blindness and a short life span, underwent right lateral sinus thrombosis in one stage. The second patient underwent sagittal and right lateral sinus thrombosis in four stages, and her remaining lateral sinus spontaneously occluded. A small untreated fistula persists in the right lower sigmoid. The third patient underwent sagittal sinus, left lateral sinus, and left parietal sinus thrombosis in six stages. A small untreated far-anterior fistula persists. All “satellite” fistulae have spontaneously disappeared. The small asymptomatic untreated residual fistulae have no demonstrable retrograde venous drainage and, therefore, require no treatment at this time.

2019 ◽  
Vol 276 (6) ◽  
pp. 1853-1854
Author(s):  
Alessandro Scorpecci ◽  
Pasquale Marsella ◽  
Sara Giannantonio ◽  
Paola Zangari ◽  
Daniela Longo ◽  
...  

2012 ◽  
Vol 2012 ◽  
pp. 1-3 ◽  
Author(s):  
P. O. Okunola ◽  
G. E. Ofovwe ◽  
M. T. Abiodun ◽  
C. P. Azunna

Cerebral venous sinus (sinovenous) thrombosis (CSVT) is a rare life-threatening disorder in childhood that is often misdiagnosed. CSVT encompasses cavernous sinus thrombosis, lateral sinus thrombosis, and superior sagittal sinus thrombosis (SSST). We present an adolescent girl who was well until two weeks earlier when she had a throbbing frontal headache and fever with chills; she later had dyspnoea, jaundice, melena stool, multiple seizures, nuchal rigidity, and monoparesis of the right lower limb a day before admission. Urine test forSalmonellatyphi Vi antigen was positive, and Widal reaction was significant. Serial cranial computerized tomography scans revealed an expanding hypodense lesion in the parafalcine region consistent with SSST or a parasagittal abscess. Inadvertent left parietal limited craniectomy confirmed SSST. She recovered completely with subsequent conservative management. Beyond neuropsychiatric complications of Typhoid fever, CSVT should be highly considered when focal neurologic deficits are present.


2009 ◽  
Vol 129 (7) ◽  
pp. 729-734 ◽  
Author(s):  
Norma de Oliveira Penido ◽  
José Ricardo Gurgel Testa ◽  
Daniel Paganini Inoue ◽  
Oswaldo Laércio Mendonça Cruz

1986 ◽  
Vol 64 (5) ◽  
pp. 724-730 ◽  
Author(s):  
Pierre Lasjaunias ◽  
Ming Chiu ◽  
Karel Ter Brugge ◽  
Atul Tolia ◽  
Michel Hurth ◽  
...  

✓ The authors describe their experience with four cases of dural arteriovenous malformation (AVM) which led them to analyze the clinical aspects of these lesions in an attempt to understand their pathophysiology. An additional 191 previously reported cases of dural AVM's were reviewed with special attention to the mechanism of intradural, central, and peripheral nervous system manifestations. Apart from the peripheral cranial nerve symptoms, which are most likely due to arterial steal, the central nervous system (CNS) symptoms appear to be related to passive venous hypertension and/or congestion. Generalized CNS symptoms can be related to cerebrospinal fluid malabsorption due either to increased pressure in the superior sagittal sinus, to venous sinus thrombosis, or to meningeal reaction resulting from minimal subarachnoid hemorrhages. These phenomena are not related to the anatomical type of venous drainage. On the other hand, focal CNS symptoms are specifically indicative of cortical venous drainage. Seizures, transient ischemic attacks, motor weakness, and brain-stem and cerebellar symptoms can be encountered depending on the territory of the draining vein or veins. Therefore, the localizing value of focal CNS symptomatology relates to the venous territory and not to the nidus or to the arterial supply characteristics of dural AVM's. Furthermore, the venous patterns of various dural AVM's at the base of the skull are expressed by differences in their clinical presentation. Dural AVM's of the floor of the anterior cranial fossa and of the tentorium are almost always drained by the cortical veins and, therefore, have a high risk of intradural bleeding. The remarkable similarities in the manifestations of dural and brain AVM's and the differences in the manifestations of dural and spinal dural AVM's are pointed out. High-quality angiograms and a multidisciplinary approach to the study of dural AVM's will provide the best understanding of their symptoms and, therefore, the most appropriate treatment strategy.


2000 ◽  
Vol 114 (11) ◽  
pp. 874-877 ◽  
Author(s):  
Grewal ◽  
Bachi T. Hathiram ◽  
Ritu Agarwal ◽  
Ashwin Dwivedi ◽  
Rohan Walvekar

Otitic hydrocephalus is characterized by increased intra – cranial pressure without focal signs of neurological dysfunction. It usually occurs secondary to lateral sinus thrombosis more commonly on the right side, but it can also occur without lateral sinus thrombosis. With the advent of new antibiotics there has been a spectacular decrease in the complications of otitis media. Otogenic intra – cranial hypertension, always an uncommon condition, is seen only very rarely nowadays. Tubercular otitis media still occurs in India, and due to delays in its diagnosis it usually presents with complications. We present three patients with otitic hydrocephalus of tubercular origin.


1995 ◽  
Vol 83 (3) ◽  
pp. 539-545 ◽  
Author(s):  
James M. Herman ◽  
Robert F. Spetzler ◽  
Joshua B. Bederson ◽  
James M. Kurbat ◽  
Joseph M. Zabramski

✓ A rat model was developed to determine the role of sinus thrombosis and elevated sinus pressures in the pathogenesis of dural arteriovenous malformations (AVMs) Five protocols were tested to compare various sinus pressures and thrombosis of a sinus: 1) Control I, sham operation (five animals); 2) Control II, occlusion of the right common carotid artery, the right external jugular vein, and the vein draining the left transverse sinus, as well as thrombosis of the sagittal sinus (10 animals); 3) arteriovenous fistula (AVF) I, anastomosis of the right common carotid artery to the external jugular vein causing retrograde flow through the transverse sinus (10 animals); 4) AVF II, anastomosis (as described in AVF I) and thrombosis of the sagittal sinus (12 animals); 5) AVF III, anastomosis (as described in AVF I) as well as thrombosis of the sagittal sinus and occlusion of the vein draining the transverse sinus on the left (12 animals). Mean arterial and sagittal sinus pressures were monitored and cerebral angiograms were obtained intraoperatively and again 90 days later. Afterward, the animals were sacrificed and their brains and dura were examined histologically. Formation of a fistula resulted in a significant (p < 0.05) threefold increase in sagittal sinus pressure in the AVF II group and a significant (p < 0.05) sixfold increase in the AVF III group. Seven dural AVMs (three in the AVF II group and four in the AVF III group) were demonstrated angiographically and histologically. The seven malformations were located adjacent to a thrombosed sagittal sinus. All lesions were within the dura and sinus wall with direct thrombus—sinus wall connections demonstrated in four of the malformations. The other three lesions displayed arteriovenous connections within the sinus wall and dura. These data suggest the importance of not only sinus thrombosis but also sinus hypertension in the development of a dural AVM.


1988 ◽  
Vol 102 (10) ◽  
pp. 877-882 ◽  
Author(s):  
K. TveteråS ◽  
S. Kristensen ◽  
H. Dommerby

AbstractThe incidence of both lateral and cavernous sinus thrombophlebitis has been significantly reduced in the antibiotic era. Since septic cavernous sinus thrombosis (CST) is mainly a complication of facial abscesses and septic lateral sinus thrombosis (LST) is almost invariably due to chronic otitis media, both conditions are of clinical relevance to the otolaryngologist.The predominant bacterium in septic CST isStaphylococcus aureuswhereas in septic LST the bacteriology is very similar to that found in chronic otitis media. The diagnosis of septic CST can be established in most cases after thorough clinical examination, and contrast computerized tomography (CT) using the coronal projection usually confirms the clinical diagnosis. The signs and clinical course of septic LST are non-specific and the final diagnosis rests upon radiological investigations including CT-scan. The treatment of both conditions consists of broad-spectrum antibiotics, including beta-lactamase resistant penicillin in cases of septic CST. Most cases of septic LST also require surgical intervention.Two cases oi septic intracranial sinus tiuombosis ate presented. The need for early diagnosis and treatment of this potentially lethal condition is emphasized.


1985 ◽  
Vol 99 (1) ◽  
pp. 91-94 ◽  
Author(s):  
F. Debruyne

AbstractIn spite of the increasing use of more efficient medical therapy, different types of otogenic intracranial complications continue to occur.A detailed description of two cases of lateral sinus thrombosis illustrates that its present-day clinical course can differ from the traditional clinical picture of the disease.


2001 ◽  
Vol 94 (1) ◽  
pp. 130-132 ◽  
Author(s):  
Cheng-Shyuan Rau ◽  
Chun-Chung Lui ◽  
Cheng-Loong Liang ◽  
Han-Jung Chen ◽  
Yeh-Lin Kuo ◽  
...  

✓ There is a wide variety of disorders associated with thrombosis of the superior sagittal sinus (SSS), including infectious disease, noninfectious conditions such as vasculitis and hypercoagulable states, and complications arising from pregnancy or use of oral contraceptive medications. Despite these well-defined associations, approximately 25% of the cases remain idiopathic. In this article the authors describe a patient who was found to have SSS thrombosis while experiencing a thyrotoxic phase of Graves disease. The patient presented with intracerebral hemorrhage, subarachnoid hemorrhage, seizure, coma, a raised fibrinogen concentration, low protein C activity, and atrial fibrillations. Thrombolysis was successfully performed despite the coexistence of thrombosis and intracranial hemorrhage. Patients with thyrotoxicosis and a diffuse goiter may be predisposed to the development of SSS thrombosis, as a result of hypercoagulation and stasis of local venous blood flow. In the present case, a patient in whom thrombosis coexisted with intracranial hemorrhage was successfully treated using thrombolytic therapy.


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