scholarly journals Intrahepatic Large Bile Duct

2020 ◽  
Author(s):  
Keyword(s):  
Bile Duct ◽  
Large Bile Duct

Total parenteral nutrition-induced cholestasis mimicking large bile duct obstruction

1982 ◽  
Vol 6 (6) ◽  
pp. 787-792 ◽  
Author(s):  
J. J. BODY ◽  
H. BLEIBERG ◽  
D. BRON ◽  
H. MAURAGE ◽  
V. BIGIRIMANA ◽  
...  
Keyword(s):  
Bile Duct ◽  
Parenteral Nutrition ◽  
Total Parenteral Nutrition ◽  
Bile Duct Obstruction ◽  
Duct Obstruction ◽  
Large Bile Duct

Delayed Endoscopic Papillary Large Balloon Dilation After Sphincterotomy for Removing Large Bile Duct Stones in Patients with Acute Cholangitis

2014 ◽  
Vol 59 (6) ◽  
pp. 1302-1306 ◽  
Author(s):  
Jong Chan Lee ◽  
Jong Ho Moon ◽  
Hyun Jong Choi ◽  
Dong Choon Kim ◽  
Moon Han Choi ◽  
...  
Keyword(s):  
Bile Duct ◽  
Acute Cholangitis ◽  
Balloon Dilation ◽  
Bile Duct Stones ◽  
Large Balloon ◽  
Large Balloon Dilation ◽  
Large Bile Duct

Large bile duct stones

ERCP ◽  
2014 ◽  
pp. 250-266
Author(s):  
Julia McNabb-Baltar ◽  
Alan Barkun
Keyword(s):  
Bile Duct ◽  
Bile Duct Stones ◽  
Large Bile Duct

scholarly journals What Are the Precursor and Early Lesions of Peripheral Intrahepatic Cholangiocarcinoma?

2014 ◽  
Vol 2014 ◽  
pp. 1-9 ◽  
Author(s):  
Yasuni Nakanuma ◽  
Akemi Tsutsui ◽  
Xiang Shan Ren ◽  
Kenichi Harada ◽  
Yasunori Sato ◽  
...  
Keyword(s):  
Bile Duct ◽  
Bile Ducts ◽  
Precursor Lesions ◽  
Intraductal Papillary Neoplasm ◽  
Combined Hepatocellular Cholangiocarcinoma ◽  
Biliary Adenofibroma ◽  
Bile Duct Lesions ◽  
Large Bile Duct ◽  
Papillary Neoplasm ◽  
Bile Duct Adenoma

Cholangiocarcinoma (CC) is divided into distal, perihilar, and intrahepatic CCs (ICCS), and are further subdivided into large bile duct ICC and peripheral ICC. In distal and perihilar CC and large duct ICC, biliary intraepithelial neoplasm (BilIN) and intraductal papillary neoplasm (IPN) have been proposed as precursor lesions. Peripheral ICC, bile duct adenoma (BDA), biliary adenofibroma (BAF), and von Meyenburg complexes (VMCs) are reportedly followed by development of ICCs. Herein, we surveyed these candidate precursor lesions in the background liver of 37 cases of peripheral ICC and controls (perihilar CC, 34 cases; hepatocellular carcinoma, 34 cases and combined hepatocellular cholangiocarcinoma, 25 cases). In the background liver of peripheral ICC, BDA and BAF were not found, but there were not infrequently foci of BDA-like lesions and atypical bile duct lesions involving small bile ducts (32.4% and 10.8%, resp.). VMCs were equally found in peripheral CCs and also control CCs. In conclusion, BDA, BAF, and VMCs are a possible precursor lesion of a minority of peripheral CCs, and BDA-like lesions and atypical bile duct lesions involving small bile ducts may also be related to the development of peripheral ICC. Further pathologic studies on these lesions are warranted for analysis of development of peripheral ICCs.

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