scholarly journals Transcortical Motor Aphasia in Recovery: A Case Report

2015 ◽  
Vol 6 ◽  
Author(s):  
Stark Jacqueline
Keyword(s):  
2014 ◽  
Vol 55 (1) ◽  
pp. 87-91 ◽  
Author(s):  
Rachel B. Katz ◽  
Clifford D. Packer

2018 ◽  
Vol 45 (6) ◽  
pp. 723-726 ◽  
Author(s):  
Pan Hu ◽  
Zhi Mao ◽  
Chao Liu ◽  
Xin Hu ◽  
Hongjun Kang ◽  
...  

BMC Neurology ◽  
2021 ◽  
Vol 21 (1) ◽  
Author(s):  
Huifang Wang ◽  
Hui Liu

Abstract Background MCA has several anomalies, such as accessory MCA, duplicated MCA and twig-like MCA, up to now all these reported anomalies were hypothesized to due to the failure in fusion of the primitive arterial network. No anomaly of over fused MCA has been reported. Case presentation A 59- year- old male was hospitalized with a history of paroxysmal slurred speech and left side headache for a week, his blood pressure was 160/80 mmHg and he manifested mild incomplete motor aphasia at the time of admission. The head and neck CTA and DSA all presented a huge and tortuous left MCA, we diagnosed it an anomaly and termed it over-fused MCA. The patient’s speech impairment and headache were relieved by controlling his blood pressure. Conclusions Such an anomaly of over-fused MCA is reported for the first time, it’s not needed to put special intervention on the anomaly of the patient temporarily, but more observation are needed.


2016 ◽  
Vol 13 (2) ◽  
pp. 109-111
Author(s):  
Sachidanand Gautam ◽  
Shyam C Meena ◽  
Bhawana Gautam

The author presents a patient who developed a subacute subdural hematoma approximately 2 weeks after normal delivery with history of postnatal convulsion and lateralizing neurological symptoms. On day 12, she started to feel a nonpostural and severe throbbing headache with motor aphasia and right hemiparesis power grade 4, she was referred to our department. Left subacute subdural hematoma was confirmed by a computed tomography scan. Physical examination revealed only mild right hemiparesis. Left burr hole trepanation was performed and this was followed by uneventful postoperative course, and it completely recovered after 4 weeks. Chronic subdural hematoma should be considered when postpartum patients who have history of ecclampsia with mild to severe, persistent, and non-postural headache.Nepal Journal of Neuroscience. Vol. 13, No. 2, 2016, Page: 109-111


2016 ◽  
Vol 06 (01) ◽  
pp. 050-054
Author(s):  
Ajaya Unnithan

A 27-year-old man had severe diffuse brain injury. The patient developed malignant right MCA territory infarction on second day. Emergency decompressive craniectomy was done. He was ventilated. He developed subdural hygroma on the opposite side that was drained. He improved slowly. He had hydrocephalus. VP shunt was done. He became conscious but dependent. PEG was done for feeding. Cranioplasty was done for syndrome of trephined. The patent improved to a state of good cognition with residual motor aphasia and left hemiplegia.Posttraumatic cerebral infarction is an indicator of poor prognosis. Vasospasm, intimal dissection, and thrombosis are the mechanisms. MCA territorial infarction is rare. Usual mechanism is impact of rotational forces on relatively fixed M1 segment on the posterior margin of lower wing of sphenoid, resulting in arterial dissection or intimal damage.


Cureus ◽  
2021 ◽  
Author(s):  
Ananth G ◽  
Gaurav Venkat Cuddapah ◽  
Amit Shukla ◽  
Ramesh Shighakolli

2020 ◽  
Vol 29 (4) ◽  
pp. 685-690
Author(s):  
C. S. Vanaja ◽  
Miriam Soni Abigail

Purpose Misophonia is a sound tolerance disorder condition in certain sounds that trigger intense emotional or physiological responses. While some persons may experience misophonia, a few patients suffer from misophonia. However, there is a dearth of literature on audiological assessment and management of persons with misophonia. The purpose of this report is to discuss the assessment of misophonia and highlight the management option that helped a patient with misophonia. Method A case study of a 26-year-old woman with the complaint of decreased tolerance to specific sounds affecting quality of life is reported. Audiological assessment differentiated misophonia from hyperacusis. Management included retraining counseling as well as desensitization and habituation therapy based on the principles described by P. J. Jastreboff and Jastreboff (2014). A misophonia questionnaire was administered at regular intervals to monitor the effectiveness of therapy. Results A detailed case history and audiological evaluations including pure-tone audiogram and Johnson Hyperacusis Index revealed the presence of misophonia. The patient benefitted from intervention, and the scores of the misophonia questionnaire indicated a decrease in the severity of the problem. Conclusions It is important to differentially diagnose misophonia and hyperacusis in persons with sound tolerance disorders. Retraining counseling as well as desensitization and habituation therapy can help patients who suffer from misophonia.


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