Budd-Chiari syndrome in a kitten, caused by membranous obstruction of the caudal vena cava

1995 ◽  
Vol 31 (6) ◽  
pp. 484-491 ◽  
Author(s):  
DK Macintire ◽  
RH Henderson ◽  
C Banfield ◽  
RP Kwapien
Keyword(s):  
Vena Cava ◽  
Small Animal ◽  
Volume Overload ◽  
Abdominal Distension ◽  
Hepatic Veins ◽  
Budd Chiari Syndrome ◽  
Chiari Syndrome ◽  
Patch Graft ◽  
Dacron Patch ◽  
Stenotic Segment

An eight-month-old, male domestic shorthair kitten developed progressive abdominal distension over a six-week period. Abdominocentesis revealed a fluid with high protein (3.8 g/dl) and moderate cellularity. Infectious, cardiovascular, and neoplastic causes of posthepatic obstruction were ruled out. Partial obstruction and stenosis of the caudal vena cava (CVC) at the level of the diaphragm were detected on a contrast venogram. Exploratory surgery revealed a fibrous, web-like membrane at the site of obstruction. Resection of the stenotic segment of the CVC was not possible because of the junction of the left hepatic veins and CVC just caudal to the lesion. A 3 by 6-mm, oval Dacron patch graft was sutured into the venotomy site. Postoperative complications included fever and leukocytosis, eosinophilic pleural effusion, and transient congestive heart failure associated with volume overload. The cat is normal 16 months after surgery, with no recurrence of ascites. This is the first reported case of Budd-Chiari syndrome (BCS) in a domestic kitten. Documented herein is the first successful treatment of BCS in a small animal using a vascular, prosthetic patch graft.

scholarly journals Acute Budd Chiari Syndrome

2014 ◽  
Vol 31 (4) ◽  
pp. 223-226
Author(s):  
AFMH Uddin ◽  
M Wareshuzzaman ◽  
RK Banik ◽  
SK Saha ◽  
NC Nath ◽  
...  
Keyword(s):  
Laboratory Data ◽  
Vena Cava ◽  
Protein S ◽  
Abdominal Distension ◽  
Hepatic Veins ◽  
Budd Chiari Syndrome ◽  
Abdominal Ultrasonography ◽  
Factor Deficiency ◽  
Chiari Syndrome ◽  
The Right

Budd-Chiari syndrome (BCS) is a rare disease in population characterized by hepatic venous outflow obstruction at a site that may vary from the level of the small hepatic veins up to the junction of the inferior vena cava with the right atrium . Here we presented a case of 35 year old male who presented with abdominal pain, abdominal distension and leg oedema . Subsequent physical examination, laboratory data, abdominal ultrasonography and duplex study all confirmed the diagnosis of BCS with Protein C , protein S , and antithrombin III deficiency . But we were unable to detect whether these factor deficiency were primary or secondary. Genetic study may be used to elucidate the cause. Whether primary or secondary, standard protocol should be used to treat Budd Chiari syndrome with these factor deficiency. DOI: http://dx.doi.org/10.3329/jbcps.v31i4.21009 J Bangladesh Coll Phys Surg 2013; 31: 223-226

Recanalization and Reconstruction of a Chronically Occluded Inferior Vena Cava Through an Existing Transjugular Intrahepatic Portosystemic Shunt in the Setting of Budd-Chiari Syndrome

2021 ◽  
pp. 153857442110020
Author(s):  
Reza Talaie ◽  
Hamed Jalaeian ◽  
Nassir Rostambeigi ◽  
Anthony Spano ◽  
Jafar Golzarian
Keyword(s):  
Inferior Vena Cava ◽  
Transjugular Intrahepatic Portosystemic Shunt ◽  
Inferior Vena ◽  
Vena Cava ◽  
Portosystemic Shunt ◽  
Hepatic Veins ◽  
Budd Chiari Syndrome ◽  
Chiari Syndrome ◽  
Intrahepatic Portosystemic Shunt ◽  
The Right

Budd-Chiari syndrome (BCS) results from the occlusion or flow reduction in the hepatic veins or inferior vena cava and can be treated with transjugular intrahepatic portosystemic shunt when hepatic vein recanalization fails.1-3 Hypercoagulable patients with primary BCS are predisposed to development of new areas of thrombosis within the TIPS shunt or IVC. This case details a patient with BCS, pre-existing TIPS extending to the right atrium, and chronic retrohepatic IVC thrombosis who underwent sharp recanalization of the IVC with stenting into the TIPS stent bridging the patient until his subsequent hepatic transplantation.

Budd-Chiari Syndrome in Childhood Secondary to Inferior Vena Caval Obstruction

PEDIATRICS ◽  
1979 ◽  
Vol 63 (5) ◽  
pp. 808-812
Author(s):  
Arvind Taneja ◽  
S. K. Mitra ◽  
P. D. Moghe ◽  
P. N. Rao ◽  
N. Samanta ◽  
...  
Keyword(s):  
Inferior Vena ◽  
Vena Cava ◽  
Hepatic Veins ◽  
Budd Chiari Syndrome ◽  
Venous Outflow ◽  
Massive Ascites ◽  
Inferior Vena Caval ◽  
Uncommon Disease ◽  
Chiari Syndrome ◽  
Hepatic Venous Outflow

Budd-Chiari syndrome is an uncommon disease caused by an obstruction to hepatic venous outflow either at the level of the hepatic veins or in the hepatic part of the inferior vena cava. Clinically, it presents with ascites, abdominal pain, hepatomegaly, edema, and occasionally jaundice. The syndrome was first recognised by Lamboran1 in 1842 and later described by Budd2 in 1846 and Chiari3 in 1899. The syndrome is caused by obstruction to the hepatic veins. In the Fig 1. Photograph showing massive ascites and dilated superficial abdominal veins. majority of cases, the obstruction is ascribed to obliterative thrombophlebitis of unknown cause.4

scholarly journals A young man with progressive jaundice and abdominal distension

2019 ◽  
Vol 2 (1) ◽  
pp. 6-8
Author(s):  
Sith Siramolpiwat ◽  
Tanabute Limprukkasem
Keyword(s):  
Abdominal Distension ◽  
Caudate Lobe ◽  
Hepatic Veins ◽  
Budd Chiari Syndrome ◽  
Complete Obliteration ◽  
Chiari Syndrome ◽  
Life Threatening ◽  
Hepatic Venous Outflow ◽  
Complete Thrombosis ◽  
Upper Abdomen

Budd-Chiari syndrome (BCS) is a rare, life-threatening disease caused by obstruction of hepatic venous outflow. Common presentations are right upper quadrant pain, ascites, and hepatomegaly. A diagnosis of Budd-Chiari syndrome should be suspected when liver disease occurs in a patient with known risk factors for hypercoagulable state. We report a 22-years old transgender man presented with jaundice, marked ascites and hepatomegaly. Imaging studies showed complete thrombosis of all hepatic veins. Finally, he was diagnosed with Budd-Chiari syndrome with antiphospholipid syndrome.   Figure 1  ภาพ CT upper abdomen แสดงให้เห็น marked ascites with heterogeneous liver enhancement, complete obliteration of all hepatic, caudate lobe hypertrophy, small intrahepatic venous collateral และ small arterial enhancing nodules

scholarly journals Balloon cavoplasty in membranous obstruction of vena cava

2017 ◽  
Vol 5 (7) ◽  
pp. 3239
Author(s):  
Sunil Abhisek B. ◽  
Kumar B.
Keyword(s):  
Right Atrium ◽  
Femoral Vein ◽  
Vena Cava ◽  
Hepatic Veins ◽  
Budd Chiari Syndrome ◽  
Case Scenario ◽  
Effective Treatment Option ◽  
Chiari Syndrome ◽  
Membranous Obstruction ◽  
Percutaneous Transluminal

A hepatic venous outflow tract obstruction at any level is considered as Budd Chiari syndrome (BCS). Primary BCS is usually due to a congenital membrane causing the obstruction; referred to as membranous obstruction of vena Cava (MOVC). In the past MOVC was predominantly treated through surgery, percutaneous transluminal balloon angioplasty (PTBA) is an alternative and effective form of treatment. Case scenario of a 32-year-old gentleman presented himself with complaints of hematemesis for one year and hematochezia for three months. Hepatomegaly was noted. An ultrasonogram revealed a dilated IVC, till its hepatic portion, and also the Hepatic Veins. There was a fibrous membrane like structure of about 5 mm thickness that was obstructing the IVC flow. Patient was taken up for venogram for conformation of diagnosis and intervention. 6F NIH catheter was introduced from Femoral vein, an injection into the IVC showed contrast not flowing into right atrium and there was a membranous obstruction for the forward flow. A Brockenbrough’s trans-septal needle with Mullins Sheath was used to puncture the membrane and right atrium was entered. A 22 mm Inouye mitral valvuloplasty balloon (Single Balloon, Toray Medical, Tokyo) was taken and positioned at the membrane and inflated and deflated several times with incremental pressures till the waist of the balloon disappeared. Conclusion membranous obstruction of vena cava (MOVC) is a common cause of primary Budd Chiari syndrome. Percutaneous transluminal angioplasty using Inoue or mansfield balloon with or without stenting is a safe and effective treatment option.

scholarly journals Acute Budd–Chiari syndrome due to a simple liver cyst

2014 ◽  
Vol 96 (1) ◽  
pp. 000-000 ◽  
Author(s):  
J Long ◽  
H Vaughan-Williams ◽  
J Moorhouse ◽  
H Sethi ◽  
N Kumar
Keyword(s):  
Liver Failure ◽  
Vena Cava ◽  
Liver Cyst ◽  
Organ Support ◽  
Hepatic Veins ◽  
Emergency Laparotomy ◽  
Budd Chiari Syndrome ◽  
Simple Cyst ◽  
Cystic Fluid ◽  
Chiari Syndrome

Simple liver cysts are common, rarely causing significant morbidity or mortality. Budd–Chiari syndrome (BCS) is caused by obstruction of hepatic venous outflow and is the leading cause of postsinusoidal liver failure. We present a rare case of BCS caused by a simple hepatic cyst. A 16cm × 16cm liver cyst was found on computed tomography of a 66-year-old woman presenting with abdominal pain. The cyst had become infected, thus enlarged, exerting mass effect with almost complete compression of the inferior vena cava. Shortly after admission, the patient developed acute liver failure, with deranged clotting and hepatic encephalopathy requiring full organ support on the intensive care unit. Cardiac output studies showed a low cardiac index of 1.4l/min/m2. An emergency laparotomy with fenestration of the cyst and drainage of 2l of purulent material led to a full recovery. Intraoperative cystic fluid aspirates later confirmed no evidence of Echinococcus. Histology confirmed a simple cyst. Liver biopsies showed severe, confluent, bridging necrosis, without background parenchymal liver disease. Acute BCS due to rapid compression of all major hepatic veins leading to fulminant hepatic failure is rare. Our case highlights a clinically significant complication of a simple liver cyst of which clinicians should be aware when managing these ‘innocent’ lesions.

Numerical Study of Blood Flow in the Hepatic Portion of the Inferior Vena Cava

2007 ◽  
Author(s):  
M. Matsubara ◽  
M. Watanabe ◽  
S. Watanabe ◽  
K. Konishi ◽  
S. Yamaguchi ◽  
...  
Keyword(s):  
Blood Flow ◽  
Inferior Vena Cava ◽  
Vessel Wall ◽  
Inferior Vena ◽  
Numerical Study ◽  
Vena Cava ◽  
Hepatic Veins ◽  
Partial Occlusion ◽  
Budd Chiari Syndrome ◽  
Chiari Syndrome

Budd-Chiari syndrome (BCS) is complete or partial occlusion in the hepatic veins and the hepatic portion of the inferior vena cava (IVC). The cause of BCS is not well known yet, however: abnormal vessel wall shear stress caused by blood flow is thought to increase the likelihood of developing BCS. In order to reveal the formation mechanism of BCS, we construct several vessel models of the IVC and hepatic veins from medical images and study the characteristics of the blood flow in the vicinity of the junctions of the hepatic veins with the IVC numerically.

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