scholarly journals Calcific tendinitis - supraspinatus tendon

2022 ◽  
Author(s):  
Bahman Rasuli
2018 ◽  
Vol 6 (2) ◽  
pp. 232596711775290 ◽  
Author(s):  
Anna Jungwirth-Weinberger ◽  
Christian Gerber ◽  
Glenn Boyce ◽  
Thorsten Jentzsch ◽  
Simon Roner ◽  
...  

Background: Passive glenohumeral range of motion may be characteristically limited to specific shoulder pathologies. While pain associated with loss of range of passive external glenohumeral rotation is recognized as a salient feature in adhesive capsulitis, restriction of glenohumeral range of motion in calcific tendinitis of the supraspinatus tendon has never been studied. Hypothesis: On the basis of clinical observation, we hypothesized that calcific tendinitis of the supraspinatus tendon is associated with loss of passive glenohumeral abduction without loss of external rotation. Study Design: Cohort study; Level of evidence, 3. Methods: Ranges of passive glenohumeral rotation and abduction, which are measured with a standardized protocol in our institution, were retrospectively reviewed and compared for patients diagnosed with either adhesive capsulitis or calcific tendinitis of the supraspinatus tendon. A total of 57 patients met the inclusion criteria for the calcific tendinitis, and 77 met the inclusion criteria for the adhesive capsulitis group. Results: When compared with the contralateral, unaffected shoulder, glenohumeral abduction in the calcific tendinitis group was restricted by a median of 10° (interquartile range [IQR], –20° to –5°) as opposed to glenohumeral external rotation, which was not restricted at all (median, 0°; IQR, 0° to 0°). The adhesive capsulitis group showed a median restriction of glenohumeral abduction of 40° (IQR, –50° to –30°) and a median restriction of passive glenohumeral external rotation of 40° (IQR, –60° to –30°). Conclusion: Calcific tendinitis of the supraspinatus does not typically cause loss of external rotation but is frequently associated with mild isolated restriction of abduction. This finding can be used to clinically differentiate adhesive capsulitis from calcific tendinitis.


2007 ◽  
Vol 42 (3) ◽  
pp. 400 ◽  
Author(s):  
Eui-Sung Choi ◽  
Kyoung-Jin Park ◽  
Yong-Min Kim ◽  
Dong-Soo Kim ◽  
Hyun-Chul Shon ◽  
...  

1976 ◽  
Vol 11 (3) ◽  
pp. 451
Author(s):  
Kwon Ick Ha ◽  
Dong Hyun Lee

2006 ◽  
Vol 218 (1) ◽  
pp. 45-46 ◽  
Author(s):  
S. Bittmann

2020 ◽  
Vol 2020 ◽  
pp. 1-3
Author(s):  
Masanori Wako ◽  
Jiro Ichikawa ◽  
Kensuke Koyama ◽  
Yoshihiro Takayama ◽  
Hirotaka Haro

Calcific tendinitis of the supraspinatus tendon in adults is common, but it is extremely rare in children. This report presents an unusual case of a 2-year-old boy with calcific tendinitis of the supraspinatus tendon. A mother brought her 2-year-old son to our hospital with a fever and severe left shoulder pain. Examination revealed a temperature of 38.6°C accompanied by a swollen shoulder with extreme pain and restricted movement. The radiographs of his left shoulder showed a large radio-opacity in the subacrominal region, and magnetic resonance imaging showed an elongated T1 and T2 hypointense signal above the supraspinatus tendon. Although these images were suggestive of calcific tendinitis of the supraspinatus tendon, we performed an open biopsy and resection in order to differentiate between a suspected diagnosis of calcific tendinitis, which is incredibly rare within pediatric patients, and infection or a soft tissue tumor. Finally, calcific tendinitis of the supraspinatus tendon was diagnosed by pathologic experiment and successfully treated, with complete resolution of pain and movement. Because only four other pediatric cases of calcific tendinitis of the supraspinatus tendon have ever been reported, there is a lack of information on the diagnostic process, management, and treatment of such a condition in young patients. Calcific tendinitis of the supraspinatus tendon still should be considered when encountering cases with typical findings even if the patient is a child.


Author(s):  
Josephina A. Vossen

Chapter 37 discusses calcific hydroxyapatite deposition disease (HADD). Hydroxyapatite (HA) crystal deposition disease is a systemic disease of unknown etiology that is caused by periarticular and/or intraarticular deposition of HA crystals. The shoulder is the most commonly involved joint with calcification in the supraspinatus tendon, but not all patients with HADD are symptomatic. Radiography is the main diagnostic tool for HADD, which may show calcifications of varying size and shape in the periarticular tendons, bursae, and joint capsule with joint destruction. Ultrasound can be useful in evaluation and image-guided treatment of calcific tendinitis.


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