Mixed secondary bacterial infection is associated with severe lesions of chromoblastomycosis in a neglected population from Brazil

2019 ◽  
Vol 95 (2) ◽  
pp. 201-207
Author(s):  
Sirlei G. Marques ◽  
Maria Rosa Q. Bomfim ◽  
Conceição de Maria P.S. Azevedo ◽  
Cleide Viviane B. Martins ◽  
Ana Claudia G. Marques ◽  
...  
Author(s):  
Mohammad M. Al-Qattan ◽  
Nada G. AlQadri ◽  
Ghada AlHayaza

Abstract Introduction Herpetic whitlows in infants are rare. Previous authors only reported individual case reports. We present a case series of six infants. Materials and Methods This is a retrospective study of six cases of herpetic whitlows in infants seen by the senior author (MMA) over the past 23 years (1995–2017 inclusive). The following data were collected: age, sex, digit involved in the hand, mode of transmission, time of presentation to the author, clinical appearance, presence of secondary bacterial infection, presence of other lesions outside the hand, method of diagnosis, treatment, and outcome. Results All six infants initially presented with classic multiple vesicles of the digital pulp. In all cases, there was a history of active herpes labialis in the mother. Incision and drainage or deroofing of the vesicles (for diagnostic purposes) resulted in secondary bacterial infection. Conclusion The current report is the first series in the literature on herpetic whitlows in infants. We stress on the mode of transmission (from the mother) and establishing the diagnosis clinically. In these cases, no need for obtaining viral cultures or polymerase chain reaction; and no medications are required. Once the vesicles are disrupted, secondary bacterial infection is frequent and a combination of oral acyclovir and intravenous antibiotics will be required.


2018 ◽  
Vol 3 (3) ◽  
Author(s):  
Wendy Kwok ◽  
Kate Charlotte Mellor

<strong>PICO question</strong><br /><p>In cats with feline acne and secondary bacterial folliculitis or furunculosis, is topical or systemic antimicrobial therapy superior for reducing time to resolution and severity of clinical signs?</p><strong>Clinical bottom line</strong><br /><p>There is no sufficient evidence to compare topical versus systemic treatment in feline acne with secondary folliculitis/furunculosis.</p><br /> <img src="https://www.veterinaryevidence.org/rcvskmod/icons/oa-icon.jpg" alt="Open Access" /> <img src="https://www.veterinaryevidence.org/rcvskmod/icons/pr-icon.jpg" alt="Peer Reviewed" />


PEDIATRICS ◽  
1972 ◽  
Vol 50 (3) ◽  
pp. 481-483
Author(s):  
John M. Neff ◽  
Robert H. Drachman

The records of all children who received smallpox vaccination in a large comprehensive care clinic were reviewed for complications. There were 944 vaccinations performed during 1968. Of these, 539 were primary vaccinations, 32 on children in the age group less than 1, and 517 on children in the age group 1 to 4. There were nine complications observed, all in children in the age group 1 to 4. Five of these complications were generalized erythematous urticarial eruptions, three were auto-inoculations, and one was a secondary bacterial infection at the site of vaccination.


PEDIATRICS ◽  
1975 ◽  
Vol 55 (2) ◽  
pp. 294-294
Author(s):  
William T. Speck

Dr. Alan M. Polson's article on gingival and periodontal problems in children was most informative.1 However, I found the last sentence somewhat puzzling, i.e., "Antibiotics are a help in preventing secondary infection." I wonder if the author could (1) provide evidence to substantiate his suggestion that secondary bacterial infection occur with acute herpetic gingivostomatitis and (2) substantiate his claim that antibiotics prevent such a complication?


2020 ◽  
Vol 68 (3) ◽  
pp. 684
Author(s):  
AriGeorge Chacko ◽  
AnanthP Abraham ◽  
MandeepSingh Bindra

2010 ◽  
Vol 7 (2) ◽  
pp. 103-114 ◽  
Author(s):  
Amanda M. Jamieson ◽  
Shuang Yu ◽  
Charles H. Annicelli ◽  
Ruslan Medzhitov

1949 ◽  
Vol 89 (1) ◽  
pp. 53-68 ◽  
Author(s):  
Carl G. Harford ◽  
Virginia Leidler ◽  
Mary Hara ◽  

1. The normal lung of the mouse possesses the power of reducing markedly its content of Type I pneumococci within 3 hours after inhalation of the organisms in the form of fine droplets. 2. Lungs with fully developed influenza viral pneumonia not only fail to reduce the pulmonary content of pneumococci administered in this manner but, on the contrary, support their growth. 3. After intrabronchial inoculation into mice, influenza virus multiplies rapidly in the lung within 24 hours. 4. Criteria have been established for distinction between true viral lesions of the lung and changes due to the inoculation of diluents as vehicles for the virus. 5. 24 hours after inoculation of virus, there are no macroscopic lesions in the lung and the microscopic changes are due to the diluent. 6. Presence and multiplication of the virus in the lung 24 hours after inoculation have no apparent effect on the power of the lung to reduce rapidly its content of inhaled pneumococci. 7. The effect of the virus in lowering resistance to secondary bacterial infection appears to be due to the presence of the lesion produced by the virus.


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