scholarly journals Effect of Entodinium caudatum on starch intake and glycogen formation by Eudiplodinium maggii in the rumen and reticulum

2017 ◽  
Vol 57 ◽  
pp. 38-49 ◽  
Author(s):  
Grzegorz Bełżecki ◽  
Neil R. McEwan ◽  
Barbara Kowalik ◽  
Tadeusz Michałowski ◽  
Renata Miltko
Keyword(s):  
Glycogen Formation

Zoned hepatic glycogen formation during refeeding

1987 ◽  
Vol 15 (4) ◽  
pp. 672-673 ◽  
Author(s):  
MARK A. JEPSON ◽  
GILLIAN M. LAWRENCE ◽  
DERYCK G. WALKER
Keyword(s):  
Hepatic Glycogen ◽  
Glycogen Formation

scholarly journals Studies on glycogen synthesis in pigeon liver homogenates. Glycogen synthesis from glucose monophosphates and uridine diphosphate glucose

1967 ◽  
Vol 105 (2) ◽  
pp. 515-519 ◽  
Author(s):  
V. N. Nigam
Keyword(s):  
Time Course ◽  
Initial Rate ◽  
Glycogen Synthesis ◽  
Uridine Diphosphate ◽  
Cytoplasmic Fraction ◽  
Uridine Diphosphate Glucose ◽  
Diphosphate Glucose ◽  
Liver Homogenates ◽  
Pigeon Liver ◽  
Glycogen Formation

Comparative time-course studies of glycogen synthesis from glucose 6-phosphate, glucose 1-phosphate and UDP-glucose show that glucose 1-phosphate forms glycogen at an initial rate faster than that obtained with glucose 6-phosphate and UDP-glucose. After 5min. the rates from glucose monophosphates are considerably slower. 2,4-Dinitrophenol decreases glycogen synthesis from both glucose monophosphates, whereas arsenate and EDTA increase glycogen synthesis from glucose 1-phosphate and inhibit the reaction from glucose 6-phosphate, galactose and galactose 1-phosphate. Mitochondria-free pigeon liver cytoplasmic fraction forms less glycogen from glucose monophosphates than does the whole homogenate. 2-Deoxyglucose 6-phosphate inhibits glycogen synthesis from glucose monophosphates. Glycogen formation from UDP-glucose is relatively unaffected by dinitrophenol, by arsenate, by EDTA, by 2-deoxyglucose 6-phosphate and by the removal of mitochondria from the whole homogenate.

scholarly journals Glycogen Formation from Glycols and their Esters in the Livers of Chicks

1972 ◽  
Vol 36 (3) ◽  
pp. 497-499
Author(s):  
Minoru YOSHIDA ◽  
Haruhisa IKUMO
Keyword(s):  
Glycogen Formation

Glycogen Formation in Rats

1930 ◽  
Vol 3 (3) ◽  
pp. 297-302 ◽  
Author(s):  
Esther M. Greisheimer ◽  
Olga H. Johnson
Keyword(s):  
Glycogen Formation

Antagonism of anethole and adrenal extract on glycogen formation in adrenalectomized rats

1951 ◽  
Vol 34 (2) ◽  
pp. 352-359
Author(s):  
Lucille Coté ◽  
Jeanne Hughes ◽  
J.J. Oleson ◽  
J.H. Williams
Keyword(s):  
Glycogen Formation ◽  
Adrenalectomized Rats

Coordinated regulation of hepatic glycogen formation in perfused rat liver by glucose and lactate

1994 ◽  
Vol 266 (4) ◽  
pp. E583-E591 ◽  
Author(s):  
Z. Zhang ◽  
J. Radziuk
Keyword(s):  
Rat Liver ◽  
Dose Response ◽  
Glycogen Synthesis ◽  
Hepatic Glycogen ◽  
Perfused Rat Liver ◽  
Response Curves ◽  
Liver Preparation ◽  
Lactate Uptake ◽  
Glucose Output ◽  
Glycogen Formation

Lactate has been found to enhance the formation of glycogen from both glucose and lactate as substrate (Z. Zhang and J. Radziuk. Biochem. J. 280: 415–419, 1991). To evaluate the relative importance of its role as substrate and regulatory factor, a dual dose-response evaluation was done by adding variable amounts of glucose and lactate to the medium in a recirculating perfused rat liver preparation. Nine groups of perfusions were performed utilizing three different levels of carbon infusion into the system: 0.25, 1.0, and 2.0 mg/min. These levels of carbon infusion were further subdivided into different relative amounts of glucose and lactate. Lactate uptake by the perfused liver was linearly related with net glucose output, regardless of the glucose concentrations. In contrast to this, the effect of lactate uptake on the rate of glycogen synthesis is saturable. Moreover, the rate of glycogen formation at which this saturation occurs is dependent only on the mean perfusate glucose concentration. The highest amount of glycogen formed in a 2-h period was 50 +/- 7 mg and the lowest 3.4 +/- 0.3 mg. A family of dose-response curves was generated describing this dual dependence of glycogen formation (both direct and gluconeogenetic pathways) on lactate and glucose.

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