scholarly journals Apoptosis induction in gastric mucous cells in vitro: lesser potency of Helicobacter pylori than Escherichia coli lipopolysaccharide, but positive interaction with ibuprofen

2006 ◽  
Vol 12 (1) ◽  
pp. 47-56 ◽  
Author(s):  
Emma Durkin ◽  
Anthony P. Moran ◽  
Peter J. Hanson
Keyword(s):  
Escherichia Coli ◽  
Helicobacter Pylori ◽  
Mucous Cells ◽  
Apoptosis Induction ◽  
Positive Interaction ◽  
Gastric Mucous ◽  
Escherichia Coli Lipopolysaccharide

Helicobacter pylori infection produces expression of secretory component in gastric mucous cells

2001 ◽  
Vol 120 (5) ◽  
pp. A708-A709
Author(s):  
T KANEKO ◽  
H OTA ◽  
M HAYAMA ◽  
K NAKAJIMA ◽  
A YOSHIZAWA ◽  
...  
Keyword(s):  
Helicobacter Pylori ◽  
Secretory Component ◽  
Helicobacter Pylori Infection ◽  
Mucous Cells ◽  
Gastric Mucous

A Novel In Vitro Infection Model of Helicobacter pylori Using Mucin-Producing Murine Gastric Surface Mucous Cells

Helicobacter ◽  
2004 ◽  
Vol 9 (4) ◽  
pp. 302-312 ◽  
Author(s):  
Tetsufumi Takahashi ◽  
Tsukasa Matsumoto ◽  
Masahiko Nakamura ◽  
Hidenori Matsui ◽  
Hiroaki Kiyohara ◽  
...  
Keyword(s):  
Helicobacter Pylori ◽  
Infection Model ◽  
Mucous Cells

Helicobacter pylori infection produces expression of a secretory component in gastric mucous cells

2000 ◽  
Vol 437 (5) ◽  
pp. 514-520 ◽  
Author(s):  
Taimei Kaneko ◽  
Hiroyoshi Ota ◽  
Masayoshi Hayama ◽  
Taiji Akamatsu ◽  
Tsutomu Katsuyama
Keyword(s):  
Helicobacter Pylori ◽  
Secretory Component ◽  
Helicobacter Pylori Infection ◽  
Mucous Cells ◽  
Gastric Mucous

Induction of Cyclooxygenase 2 by Escherichia coli but not Helicobacter pylori lipopolysaccharide in gastric epithelial cells in vitro

Helicobacter ◽  
2003 ◽  
Vol 8 (5) ◽  
pp. 513-520 ◽  
Author(s):  
Geoff V. Smith ◽  
Anthony P. Moran ◽  
Mona Bajaj-Elliott ◽  
Michael J.G. Farthing
Keyword(s):  
Escherichia Coli ◽  
Helicobacter Pylori ◽  
Epithelial Cells ◽  
Cyclooxygenase 2 ◽  
Gastric Epithelial Cells

scholarly journals RAPID BREAKING OF TOLERANCE AGAINST ESCHERICHIA COLI LIPOPOLYSACCHARIDE IN VIVO AND IN VITRO

1972 ◽  
Vol 135 (4) ◽  
pp. 850-859 ◽  
Author(s):  
Olof Sjöberg
Keyword(s):  
Escherichia Coli ◽  
Immune Responsiveness ◽  
Spleen Cells ◽  
E Coli ◽  
Normal Spleen ◽  
Escherichia Coli Lipopolysaccharide ◽  
Tolerant State

The breaking of tolerance against the lipopolysaccharide from E. coli 055:B5 was studied. It was found that immune responsiveness recovered very slowly in vivo, tolerance still existing 3 wk after the last tolerizing injection. However, if spleen cells from tolerant mice were transferred into irradiated syngeneic recipients, the tolerant state was readily broken. Spleen cells transferred 3 days after the last tolerance-maintaining dose did not respond, whereas cells transferred on day 5 or 7 responded equally well as normal spleen cells. It was also possible to break tolerance by incubating tolerant spleen cells, which did not respond after transfer, for 20 hr in vitro before transfer into irradiated recipients. The results suggest that there exist reversibly inactivated cells in tolerant animals and that these cells can be reactivated upon removal of the cells to a neutral environment.

scholarly journals Helicobacter pylori DnaB helicase can bypass Escherichia coli DnaC function in vivo

2005 ◽  
Vol 389 (2) ◽  
pp. 541-548 ◽  
Author(s):  
Rajesh K. Soni ◽  
Parul Mehra ◽  
Gauranga Mukhopadhyay ◽  
Suman Kumar Dhar
Keyword(s):  
Escherichia Coli ◽  
Helicobacter Pylori ◽  
Temperature Sensitive ◽  
Chromosomal Dna ◽  
E Coli ◽  
Dnab Helicase ◽  
H Pylori

In Escherichia coli, DnaC is essential for loading DnaB helicase at oriC (the origin of chromosomal DNA replication). The question arises as to whether this model can be generalized to other species, since many eubacterial species fail to possess dnaC in their genomes. Previously, we have reported the characterization of HpDnaB (Helicobacter pylori DnaB) both in vitro and in vivo. Interestingly, H. pylori does not have a DnaC homologue. Using two different E. coli dnaC (EcdnaC) temperature-sensitive mutant strains, we report here the complementation of EcDnaC function by HpDnaB in vivo. These observations strongly suggest that HpDnaB can bypass EcDnaC activity in vivo.

Helicobacter pylori infection produces expression of secretory component in gastric mucous cells

2001 ◽  
Vol 120 (5) ◽  
pp. A708-A709
Author(s):  
Taimei Kaneko ◽  
Hiroyoshi Ota ◽  
Masayoshi Hayama ◽  
Kosei Nakajima ◽  
Akihiko Yoshizawa ◽  
...  
Keyword(s):  
Helicobacter Pylori ◽  
Secretory Component ◽  
Helicobacter Pylori Infection ◽  
Mucous Cells ◽  
Gastric Mucous
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