The current pathogens and treatment of hospital-acquired pneumonia/ventilator-associated pneumonia in medical intensive care units- A prospective cohort observational study

2016 ◽  
Author(s):  
Youjin Chang ◽  
Young-Jae Cho ◽  
Sang-Min Lee ◽  
Kyeongman Jeon ◽  
Seok Chan Kim ◽  
...  
Keyword(s):  
Intensive Care ◽  
Observational Study ◽  
Intensive Care Units ◽  
Prospective Cohort ◽  
Ventilator Associated Pneumonia ◽  
Hospital Acquired Pneumonia ◽  
Medical Intensive Care ◽  
Hospital Acquired

scholarly journals Etiology, incidence and mortality in patients with ventilator-associated pneumonia in adult general surgery and cardiac surgery intensive care units in University Hospital Dubrava

2020 ◽  
Vol 39 (4) ◽  
pp. 124-128
Author(s):  
Andrea Karin ◽  
Andrej Šribar ◽  
Marko Pražetina ◽  
Katerina Bakran ◽  
Jasminka Peršec
Keyword(s):  
Cardiac Surgery ◽  
Intensive Care ◽  
Intensive Care Units ◽  
General Surgery ◽  
University Hospital ◽  
Ventilator Associated Pneumonia ◽  
Timely Initiation ◽  
Incidence And Mortality ◽  
Hospital Acquired Pneumonia ◽  
Hospital Acquired

Ventilator-associated pneumonia (VAP) and hospital acquired pneumonia (HAP) strongly contribute to morbidity and mortality in intensive care units. Hospital acquired pneumonia (HAP) is pneumonia occurring 48 hours upon admission and appears not to be incubating at the time of admission. Ventilator-associated pneumonia (VAP) is a type of HAP developing in intubated patients after more than 48 hours upon mechanical ventilation. HAP and VAP are common and serious complications present in hospitalized patients. Since the diagnosis of VAP and HAP are rarely documented, we wanted to assess the incidence of VAP in General Surgery and Cardiac Surgery Intensive Care Units in 2018 and analyse the patients and procedures related factors. Patients intubated and ventilated more than 96 hours during 2018 were included. Our findings have shown that incidence of VAP in two analysed ICUs in UH Dubrava is in line with VAP incidence found in literature due to successful preventive strategies and timely initiation of antimicrobial therapy and other adjunctive procedures.

Hospital-Acquired Pneumonia in the Intensive Care Units of Polish Hospitals

2006 ◽  
Vol 27 (7) ◽  
pp. 784-786
Author(s):  
Jadwiga Wójkowska-Mach ◽  
Małgorzata Bulanda ◽  
Anna Różańska ◽  
Piotr Kochan ◽  
Piotr B. Heczko
Keyword(s):  
Escherichia Coli ◽  
Intensive Care ◽  
Mortality Rate ◽  
Intensive Care Units ◽  
Artificial Ventilation ◽  
Ventilator Associated Pneumonia ◽  
Hospital Acquired Pneumonia ◽  
Hospital Acquired ◽  
Epidemiological Characteristics ◽  
Overall Mortality

We analyzed the epidemiological characteristics of pneumonia in intensive care units of Polish hospitals. Among 11,587 patients, there were 191 cases of hospital-acquired pneumonia (HAP). The incidence of HAP was 5.6%, and that of ventilator-associated pneumonia (VAP) was 17.9%. The overall mortality rate was 12.6%, and the mortality rate for patients who received artificial ventilation was 15.0%. The predominant organisms causing HAP and VAP were Pseudomonas aeruginosa and Escherichia coli, and 21.1% of Staphylococcus aureus isolates were resistant to methicillin.

Hospital-Acquired Pneumonia in the Intensive Care Units of Polish Hospitals

2006 ◽  
Vol 27 (7) ◽  
pp. 784-786 ◽  
Author(s):  
Jadwiga Wójkowska-Mach ◽  
Małgorzata Bulanda ◽  
Anna Różańska ◽  
Piotr Kochan ◽  
Piotr B. Heczko
Keyword(s):  
Escherichia Coli ◽  
Intensive Care ◽  
Mortality Rate ◽  
Intensive Care Units ◽  
Artificial Ventilation ◽  
Ventilator Associated Pneumonia ◽  
Hospital Acquired Pneumonia ◽  
Hospital Acquired ◽  
Epidemiological Characteristics ◽  
Overall Mortality

We analyzed the epidemiological characteristics of pneumonia in intensive care units of Polish hospitals. Among 11,587 patients, there were 191 cases of hospital-acquired pneumonia (HAP). The incidence of HAP was 5.6%, and that of ventilator-associated pneumonia (VAP) was 17.9%. The overall mortality rate was 12.6%, and the mortality rate for patients who received artificial ventilation was 15.0%. The predominant organisms causing HAP and VAP were Pseudomonas aeruginosa and Escherichia coli, and 21.1% of Staphylococcus aureus isolates were resistant to methicillin.

Critical care in Colombia: Differences between teaching and nonteaching intensive care units. A prospective cohort observational study

2012 ◽  
Vol 27 (1) ◽  
pp. 104.e9-104.e17 ◽  
Author(s):  
Sandra Rubiano ◽  
Fabian Gil ◽  
Edgar Celis-Rodriguez ◽  
Henry Oliveros ◽  
Gabriel Carrasquilla
Keyword(s):  
Critical Care ◽  
Intensive Care ◽  
Observational Study ◽  
Intensive Care Units ◽  
Prospective Cohort

Hospital-Acquired Pneumonia Developed in Non-Intensive Care Units

Respiration ◽  
2009 ◽  
Vol 78 (4) ◽  
pp. 416-422 ◽  
Author(s):  
Ebru Cakir Edis ◽  
Osman N. Hatipoglu ◽  
Ilker Yilmam ◽  
Alper Eker ◽  
Ozlem Tansel ◽  
...  
Keyword(s):  
Intensive Care ◽  
Intensive Care Units ◽  
Hospital Acquired Pneumonia ◽  
Hospital Acquired

scholarly journals Disease Risk and Mortality Prediction in Intensive Care Patients with Pneumonia. Australian and New Zealand Practice in Intensive Care (ANZPIC II)

2005 ◽  
Vol 33 (1) ◽  
pp. 101-111 ◽  
Author(s):  
R. J. Boots ◽  
J. Lipman ◽  
R. Bellomo ◽  
D. Stephens ◽  
R. F. Heller
Keyword(s):  
Intensive Care ◽  
New Zealand ◽  
Clinical Signs ◽  
Community Acquired Pneumonia ◽  
Ventilator Associated Pneumonia ◽  
Invasive Ventilation ◽  
Non Invasive ◽  
Non Invasive Ventilation ◽  
Hospital Acquired Pneumonia ◽  
Hospital Acquired

This study of ventilated patients investigated pneumonia risk factors and outcome predictors in 476 episodes of pneumonia (48% community-acquired pneumonia, 24% hospital-acquired pneumonia, 28% ventilator-associated pneumonia) using a prospective survey in 14 intensive care units within Australia and New Zealand. For community acquired pneumonia, mortality increased with immunosuppression (OR 5.32, CI 95% 1.58–17.99, P<0.01), clinical signs of consolidation (OR 2.43, CI 95% 1.09–5.44, P=0.03) and Sepsis-Related Organ Failure Assessment (SOFA) scores (OR 1.19, CI 95% 1.08–1.30, P<0.001) but improved if appropriate antibiotic changes were made within three days of intensive care unit admission (OR 0.42, CI 95% 0.20–0.86, P=0.02). For hospital-acquired pneumonia, immunosuppression (OR 6.98, CI 95% 1.16–42.2, P=0.03) and non-metastatic cancer (OR 3.78, CI 95% 1.20–11.93, P=0.02) were the principal mortality predictors. Alcoholism (OR 7.80, CI 95% 1.20–17.50, P<0.001), high SOFA scores (OR 1.44, CI 95% 1.20–1.75, P=0.001) and the isolation of “high risk” organisms including Pseudomonas aeruginosa, Acinetobacter spp, Stenotrophomonas spp and methicillin resistant Staphylococcus aureus (OR 4.79, CI 95% 1.43–16.03, P=0.01), were associated with increased mortality in ventilator-associated pneumonia. The use of non-invasive ventilation was independently protective against mortality for patients with community-acquired and hospital-acquired pneumonia (OR 0.35, CI 95% 0.18–0.68, P=0.002). Mortality was similar for patients requiring both invasive and non-invasive ventilation and non-invasive ventilation alone (21% compared with 20% respectively, P=0.56). Pneumonia risks and mortality predictors in Australian and New Zealand ICUs vary with pneumonia type. A history of alcoholism is a major risk factor for mortality in ventilator-associated pneumonia, greater in magnitude than the mortality effect of immunosuppression in hospital-acquired pneumonia or community-acquired pneumonia. Non-invasive ventilation is associated with reduced ICU mortality. Clinical signs of consolidation worsen, while rationalising antibiotic therapy within three days of ICU admission improves mortality for community-acquired pneumonia patients.

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