scholarly journals TRAUBE WAVES AND MAYER WAVES

1919 ◽  
Vol 12 (3) ◽  
pp. 227-229 ◽  
Author(s):  
W. D. Halliburton
Keyword(s):  
2000 ◽  
Vol 39 (02) ◽  
pp. 118-121 ◽  
Author(s):  
S. Akselrod ◽  
S. Eyal

Abstract:A simple nonlinear beat-to-beat model of the human cardiovascular system has been studied. The model, introduced by DeBoer et al. was a simplified linearized version. We present a modified model which allows to investigate the nonlinear dynamics of the cardiovascular system. We found that an increase in the -sympathetic gain, via a Hopf bifurcation, leads to sustained oscillations both in heart rate and blood pressure variables at about 0.1 Hz (Mayer waves). Similar oscillations were observed when increasing the -sympathetic gain or decreasing the vagal gain. Further changes of the gains, even beyond reasonable physiological values, did not reveal another bifurcation. The dynamics observed were thus either fixed point or limit cycle. Introducing respiration into the model showed entrainment between the respiration frequency and the Mayer waves.


2020 ◽  
Vol 15 (10) ◽  
pp. 1821 ◽  
Author(s):  
MichaelGeorge Zaki Ghali ◽  
GeorgeZaki Ghali ◽  
EmilZaki Ghali
Keyword(s):  

1992 ◽  
Vol 15 (10) ◽  
pp. 601-605 ◽  
Author(s):  
T. Yambe ◽  
S. Nitta ◽  
Y. Katahira ◽  
T. Sonobe ◽  
S. Naganuma ◽  
...  

To assess the effect of a total artificial heart (TAH) on the autonomic nervous system a power spectral analysis of the hemodynamics in a TAH animal was done by the maximum entropy method. Two pneumatically driven sac-type ventricular assist devices were implanted as total biventricular bypass (BVB) in adult mongrel dogs to compare the differences between natural heart and TAH. Once the BVB was pumping, the natural heart was electrically fibrillated to constitute the BVB-type TAH model. In the arterial pressure waveform in animals with TAH, respiratory waves were not changed (97.7±24.6%) though Mayer waves were significantly decreased (47.5 ± 22.6%) compared with the animal with a natural heart. These results suggest that prosthetic hemodynamics in the TAH animal affect fluctuations in the cardiovascular system.


2001 ◽  
Vol 91 (1-2) ◽  
pp. 62-75 ◽  
Author(s):  
Christopher W Myers ◽  
Michael A Cohen ◽  
Dwain L Eckberg ◽  
J.Andrew Taylor

2001 ◽  
Vol 20 (2) ◽  
pp. 92-100 ◽  
Author(s):  
S.R. Seydnejad ◽  
R.I. Kitney

EP Europace ◽  
2003 ◽  
Vol 4 (Supplement_2) ◽  
pp. B113-B113
Author(s):  
J. Simek ◽  
D. Wichterle ◽  
J. Kofranek ◽  
S. Svacina

2000 ◽  
Vol 279 (4) ◽  
pp. R1257-R1267 ◽  
Author(s):  
Delphine Bertram ◽  
Christian Barrès ◽  
Yong Cheng ◽  
Claude Julien

This study examined the effect of norepinephrine reuptake blockade with desipramine (DMI) on the spontaneous variability of the simultaneously recorded arterial pressure (AP) and renal sympathetic nerve activity (SNA) in conscious rats. Acute DMI administration (2 mg/kg iv) depressed AP Mayer waves (∼0.4 Hz) and increased low-frequency (<0.2 Hz) components of AP variability. DMI decreased renal SNA variability, especially due to the abolition of oscillations related to Mayer waves. To examine whether DMI-induced changes in AP and renal SNA variabilities could be explained by alterations in the dynamic characteristics of the baroreceptor reflex loop, the frequency responses of mean AP to aortic depressor nerve stimulation were studied in urethan-anesthetized rats. DMI accentuated the low-pass filter properties of the transfer function without significantly altering the fixed time delay. The frequency responses of iliac vascular conductance to stimulation of the lumbar sympathetic chain were studied in an additional group of anesthetized rats. DMI did not markedly alter the low-pass filter properties of the transfer function and slightly increased the fixed time delay. These results suggest that the DMI-induced decrease in the dynamic gain of the baroreceptor reflex is responsible for the decreased spontaneous renal SNA variability and the accompanying increased AP variability. The “slowing down” of baroreflex responses cannot be attributed to an effect of DMI at the vascular neuroeffector junction.


2013 ◽  
Vol 27 (7) ◽  
pp. 2564-2572 ◽  
Author(s):  
Bernard Cohen ◽  
Giorgio P. Martinelli ◽  
Theodore Raphan ◽  
Adam Schaffner ◽  
Yongqing Xiang ◽  
...  

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