Erythematous plaques on the skin folds

BMJ ◽  
2021 ◽  
pp. e068621
Author(s):  
Bo Feng ◽  
Jiu-Hong Li
Keyword(s):  
Erythematous Plaques

Erythematous plaques with zosteriform distribution in a patient treated with TNF‐α inhibitor adalimumab for psoriasis

2021 ◽  
Author(s):  
Cristina Garcia‐Melendo ◽  
Xavier Cubiró ◽  
Carla Tubau ◽  
Anna Mozos ◽  
Carme Muñoz ◽  
...  
Keyword(s):  
Tnf Α ◽  
Erythematous Plaques

Enteropathic acrodermatitis in children

2021 ◽  
Vol 11 (2) ◽  
pp. 21-28
Author(s):  
V.P. Novikova ◽  
◽  
A.A. Pokhlebkina ◽  
D.V. Zaslavsky ◽  
A.I. Khavkin ◽  
...  
Keyword(s):  
Zinc Deficiency ◽  
Topical Therapy ◽  
Clinical Manifestations ◽  
Skin Lesions ◽  
Knee Joints ◽  
Zinc Metabolism ◽  
Plasma Zinc ◽  
Significant Treatment ◽  
Zinc Concentrations ◽  
Erythematous Plaques

Enteropathic acrodermatitis is a rare hereditary form of zinc deficiency, characterized by periorial and acral dermatitis, alopecia and diarrhea. Refers to congenital disorders of zinc metabolism, inherited as an autosomal recessive disease resulting from mutations in the gene for the zinc transporter SLC39A4. The prevalence ranges from 1 to 9:1,000,000, with an overall incidence of 1:500,000 newborns. The disease usually manifests itself in infancy, within a few weeks of stopping breastfeeding and switching the baby to a cow's milk-based formula, or in the first days of life if artificially fed from birth. The classical clinical manifestations of acrodermatitis enteropathic are characterized by the triad: acral and periofital dermatitis, alopecia and diarrhea, but all three signs together occur only in 20% of cases. Diarrhea may develop concurrently with skin symptoms, may precede or occur later. Characteristic signs of skin lesions include sharply demarcated, dry, scaly erythematous plaques or edematous foci with vesicles and pustules on the skin of the elbow and knee joints, distal extremities, genitals, in the inguinal folds, which are usually symmetrically distributed, have sharp boundaries and irregular outlines. The course of the skin syndrome is long, as it progresses, non-healing erosive and ulcerative areas appear. Plasma zinc deficiency is the gold standard for diagnosis. Most infants with AE have low plasma zinc concentrations (<500 mcg/L or <50 mcg/dl), but a level of less than 70 mcg/L on an empty stomach or less than 65 mcg/dl in older non-dieting children is considered diagnostically significant. Treatment for this disease usually includes enteral or parenteral zinc administration, at a dose of 1-3 mg/kg/day. for elemental zinc. A clinical response is observed within 5–10 days. Supportive zinc therapy is necessary throughout the patient's life, although periods of remission have been reported. Topical therapy is also used: Dexpanthenol in the form of a cream, applied 3 times a day in the area of dermatitis, can enhance re-epithelialization. There is no significant evidence of improvement with topical zinc application. No activity restrictions are required for patients with acrodermatitis enteropathic. Key words: zinc deficiency, enteropathic acrodermatitis, children

scholarly journals Histiocytoid Sweet's syndrome presenting with annular erythematous plaques

2016 ◽  
Vol 91 (5 suppl 1) ◽  
pp. 154-156
Author(s):  
Renata Marcarini ◽  
Raquel Nardelli de Araujo ◽  
Monisa Martins Nóbrega ◽  
Karina Bittencourt Medeiros ◽  
Alexandre Carlos Gripp ◽  
...  
Keyword(s):  
Sweet’S Syndrome ◽  
Sweet's Syndrome ◽  
Erythematous Plaques

scholarly journals Erythroplasia of Queyrat Treated with 5% Imiquimod Cream — Case Report Emphasizing the Role of Human Papilloma Virus Testing in a Clinical Setting

2017 ◽  
Vol 2 (1) ◽  
pp. 83-85
Author(s):  
Anca Chiriac ◽  
Piotr Brzezinski ◽  
Cristian Podoleanu ◽  
Simona Stolnicu
Keyword(s):  
Punch Biopsy ◽  
Imiquimod Cream ◽  
Once Daily ◽  
Public Health Burden ◽  
Inflammatory Conditions ◽  
Papillomavirus Infection ◽  
Compliant Patient ◽  
Erythroplasia Of Queyrat ◽  
Erythematous Plaques

AbstractBackground:Anogenital premalignancies and malignancies often affect females and males, and human papillomavirus infection plays a crucial role in their etiopathogenesis. These lesions are very important and represent an immense public health burden.Case presentation:A 78-year-old Caucasian male presented to the Dermatology Unit for persistent, slowly progressing, well-demarcated, erythematous plaques on the glans penis, observed by the patient 18 months prior to the consultation. Variable topical treatments were applied, with no improvement and with the denial of a punch biopsy. A clinical diagnosis of erythroplasia of Queyrat was established and the test for HPV revealed an association with subtype 16 (which excluded other benign inflammatory conditions). Positive results were obtained after 4 weeks of topical application of 5% imiquimod cream, once daily, 5 times a week.Conclusion:Erythroplasia of Queyrat should be diagnosed in a non-compliant patient based on the clinical picture and HPV testing even in the absence of a biopsy, and a non-surgical treatment should be initiated immediately.

Sweet Syndrome and Pemphigus Vulgaris

2012 ◽  
Vol 16 (2) ◽  
pp. 128-130 ◽  
Author(s):  
Ashley O'toole ◽  
Maureen O'malley
Keyword(s):  
Clinical Presentation ◽  
Pemphigus Vulgaris ◽  
Neutrophilic Dermatosis ◽  
Rare Presentation ◽  
Sweet Syndrome ◽  
Autoimmune Blistering Disease ◽  
Mucous Membranes ◽  
Blistering Disease ◽  
Erythematous Plaques ◽  
New Onset

Background: Pemphigus vulgaris is an autoimmune blistering disease of the skin and mucous membranes. Sweet syndrome is an uncommon reactive neutrophilic dermatosis. Objective: This case report describes the rare presentation of a 58-year-old female patient with newly diagnosed pemphigus vulgaris who later presented with erythematous plaques and leukocytosis highly suggestive of Sweet syndrome. Methods: Review of the literature using the PubMed and Medline databases. Results: The clinical presentation of new-onset Sweet syndrome in a patient with pemphigus has been reported in the literature only on one other occasion. Conclusion: The observation of an association between Sweet syndrome and autoimmune diseases, such as pemphigus vulgaris, may ultimately lead to better understanding of the pathophysiology of this disease.

Bizarre extensive erythematous plaques on the abdomen

2018 ◽  
Vol 49 (1) ◽  
pp. 39-42
Author(s):  
Sidharth Tandon ◽  
Surabhi Sinha ◽  
Jasmeet Singh
Keyword(s):  
Case Report ◽  
Rare Case ◽  
Rare Case Report ◽  
History Of ◽  
Erythematous Plaques

We present a rare case report of a 28-year-old man with a five-month history of bizarre extensive erythematous plaques on the abdomen extending in a dermatomal fashion to the left upper back.

scholarly journals Hydrochlorothizide-induced acute generalised exanthematous pustulosis presenting with bilateral periorbital impetigo

2019 ◽  
Vol 12 (2) ◽  
pp. bcr-2017-223528 ◽  
Author(s):  
Leo E Reap ◽  
Cassandra Rodd ◽  
Jose Larios ◽  
Michael Marshall
Keyword(s):  
Adverse Reaction ◽  
Case Report ◽  
Cutaneous Adverse Reaction ◽  
Common Causes ◽  
Erythematous Plaques ◽  
Exanthematous Pustulosis

Acute generalised exanthematous pustulosis (AGEP) is a severe cutaneous adverse reaction characterised by the appearance of erythematous plaques and papules with overlying non-follicular pinpoint pustules. Drugs are the cause of AGEP in approximately 90% of cases. The most common causes include anti-infective agents (aminopenicillins, quinolones, antibacterial sulfonamides and terbinafine), antimalarials and diltiazem. To the best of our knowledge, to date there has only been one report of hydrochlorothiazide-induced AGEP. There has never been a case report of losartan-induced AGEP. Here, we present a case of AGEP that is the second case purportedly caused by hydrochlorothiazide.

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