Very Low and High Blood Viscosity are Risk Factors for Internal Flow Choking Causing Asymptomatic Cardiovascular Disease

Herein, we established the proof of the concept of internal flow choking in CVS causing cardiovascular risk through the closed-form analytical, in vitro and in silico methods. An over dose of blood-thinning drug will enhance the Reynolds number, which creates high turbulence level causing an augmented boundary layer blockage factor leading to an early undesirable biofluid/Sanal flow choking at a critical blood-pressure-ratio (BPR). The fact is that in nanoscale vessels when the pressure of fluid increases, average-mean-free-path decreases and thus, the Knudsen number reduces. It leads to the physical situation of no-slip boundary condition with compressible-viscous flow effect. Sanal-flow-choking is a compressible-viscous flow effect establishing a physical condition of the sonic-fluid-throat, at a critical blood pressure ratio (BPR). We concluded that asymptomatic-hemorrhage (AH) and acute-heart-failure (AHF) are transient-events as a result of internal flow-choking in nanoscale and/or large vessels followed by the shock wave creation and transient pressure-overshoot. We concluded that cardiovascular risk could be reduced by simultaneously lessening the blood-viscosity and flow turbulence by increasing thermal-tolerance-level in terms of BHCR and/or by decreasing the blood pressure (BP) ratio.